K. O. Aley

2.5k citations
18 papers · 2.3k · h-index 14

Impact in

Papers in

K. O. Aley

18 papers receiving 2.2k citations

Peers

K. O. Aley
Comparison fields: 5 of 93
  • Physiology 1.6k
  • Sensory Systems 254
  • Cellular and Molecular Neuroscience 914
  • Behavioral Neuroscience 83
  • Anesthesiology and Pain Medicine 97
Replace Jon D. Levine with:
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K. O. Aley relative to Jon D. Levine United States Jon D. Levine's profile →
Citations per field
00.5×1.5×
Jon D. Levine · 1×
Citations per year

Countries citing papers authored by K. O. Aley

Since Specialization
Citations

This map shows the geographic impact of K. O. Aley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. O. Aley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. O. Aley more than expected).

Fields of papers citing papers by K. O. Aley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K. O. Aley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. O. Aley. The network helps show where K. O. Aley may publish in the future.

Co-authors

The 22 scholars most cited alongside K. O. Aley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with K. O. Aley Line = papers co-authored together K. O. Aley links everyone, so they are left out of the graph.

All Works

18 of 18 papers shown
#Work
1 1999404
2 2000294
3 1998235
4 1999232
5 1999230
6 2001171
7 1996163
8 1997152
9 200294
10 200190
11 199756
12 199742
13 200141
14 200322
15
GABAergic agents-induced antinociceptive effect in mice.
198913
16
Effect of baclofen, a GABAB-agonist, on forced swimming-induced immobility in mice.
199110
17 19962
18
Studies on meperidine-induced catalepsy in mice.
19871

About K. O. Aley

K. O. Aley is a scholar working on Physiology, Cellular and Molecular Neuroscience, Molecular Biology, Neurology and Endocrinology, Diabetes and Metabolism, having authored 18 papers that have together received 2.3k indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (16 papers), Neuropeptides and Animal Physiology (8 papers), Receptor Mechanisms and Signaling (4 papers), Ion channel regulation and function (3 papers), Hormonal and reproductive studies (3 papers), Neuroscience and Neuropharmacology Research (3 papers), Botulinum Toxin and Related Neurological Disorders (3 papers) and Neurobiology and Insect Physiology Research (2 papers). The work is most often cited by research in Physiology (1.6k citations), Sensory Systems (254 citations), Cellular and Molecular Neuroscience (914 citations), Behavioral Neuroscience (83 citations) and Anesthesiology and Pain Medicine (97 citations). K. O. Aley has collaborated with scholars based in United States, India and Russia. Frequent co-authors include Jon D. Levine, Jon D. Levine, Robert O. Messing, Gordon McCarter, Robert W. Gear, Daria Mochly‐Rosen, Annick Martin, David B. Reichling, William M. Isenberg and Janine Mok. Their work appears in journals such as Journal of Neuroscience, Neuroscience, Experimental Brain Research, European Journal of Neuroscience and Neuron.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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