Jun Kuai

739 citations
16 papers · 472 · h-index 10

Impact in

    • Cancer, Hypoxia, and Metabolism
    • Cancer, Lipids, and Metabolism
    • NF-κB Signaling Pathways
  • Physiology top 10%
    • Calcium signaling and nucleotide metabolism

Papers in

Jun Kuai

15 papers receiving 465 citations

Peers

Jun Kuai
Comparison fields: 5 of 74
  • Cancer Research 133
  • Physiology 39
  • Cell Biology 123
  • Clinical Biochemistry 35
  • Molecular Biology 295
Replace David Schlütermann with:
David Schlütermann Germany
Fred Lozy United States
Caisheng Lu United States
Peta Wood Australia
Zhaoyue He Switzerland
Donald D. Anderson United States
Sylvie Carmona France
Vitaly I. Pozdeev Germany
Isabelle de Mendez United States
Paola Giglio Italy
Jun Kuai relative to David Schlütermann Germany David Schlütermann's profile →
Citations per field
00.5×3.1×
David Schlütermann · 1×
Citations per year

Countries citing papers authored by Jun Kuai

Since Specialization
Citations

This map shows the geographic impact of Jun Kuai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun Kuai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun Kuai more than expected).

Fields of papers citing papers by Jun Kuai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun Kuai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun Kuai. The network helps show where Jun Kuai may publish in the future.

Co-authors

The 25 scholars most cited alongside Jun Kuai, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jun Kuai Line = papers co-authored together Jun Kuai links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 2019122
2 201061
3 199960
4 201751
5 200348
6 200039
7 200025
8 201524
9 200418
10 200010
11 20025
12 20083
13 20213
14 20232
15 20231
16 20220

About Jun Kuai

Jun Kuai is a scholar working on Molecular Biology, Immunology, Oncology, Cell Biology and Cancer Research, having authored 16 papers that have together received 472 indexed citations. Recurring topics across this work include Cellular transport and secretion (5 papers), Calcium signaling and nucleotide metabolism (3 papers), Immune Response and Inflammation (3 papers), NF-κB Signaling Pathways (3 papers), Cytokine Signaling Pathways and Interactions (2 papers), Protein Degradation and Inhibitors (2 papers), Cancer Immunotherapy and Biomarkers (2 papers) and Pancreatic function and diabetes (1 paper). The work is most often cited by research in Cancer Research (133 citations), Physiology (39 citations), Cell Biology (123 citations), Clinical Biochemistry (35 citations) and Molecular Biology (295 citations). Jun Kuai has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Richard Kahn, Annette L. Boman, Xinjun Zhu, Wei Jia, Byron DeLaBarre, Silvana Leit, Salma B. Rafi, H. James Harwood, Liang Tong and Éric Therrien. Their work appears in journals such as Journal of Biological Chemistry, Blood, The Journal of Immunology, FEBS Letters and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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