Judith E. Unterlass

644 citations
7 papers · 121 · h-index 6

Impact in

Papers in

    • Biochemical and Molecular Research 4
    • Metabolism, Diabetes, and Cancer 1
    • ATP Synthase and ATPases Research 1
    • Protein Kinase Regulation and GTPase Signaling 1
    • Amino Acid Enzymes and Metabolism 2

Judith E. Unterlass

6 papers receiving 120 citations

Peers

Judith E. Unterlass
Comparison fields: 5 of 43
  • Biochemistry 34
  • Cancer Research 59
  • Molecular Biology 93
  • Clinical Biochemistry 8
  • Physiology 4
Replace Martin Henriksson with:
Martin Henriksson Sweden
Karen Cheng United States
Machiko Irie Japan
Chelsea K. Thornburg United States
Vanja Panic United States
Adam Rosenzweig United States
Makiko Nishi Japan
Anke Vandekeere Belgium
Yulia Panina Russia
Mengyun Xu China
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Citations per field
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Citations per year

Countries citing papers authored by Judith E. Unterlass

Since Specialization
Citations

This map shows the geographic impact of Judith E. Unterlass's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Judith E. Unterlass with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Judith E. Unterlass more than expected).

Fields of papers citing papers by Judith E. Unterlass

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Judith E. Unterlass. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Judith E. Unterlass. The network helps show where Judith E. Unterlass may publish in the future.

Co-authors

The 25 scholars most cited alongside Judith E. Unterlass, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Judith E. Unterlass Line = papers co-authored together Judith E. Unterlass links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 201629
2 201926
3 201724
4 202021
5 202014
6 20187
7 20240

About Judith E. Unterlass

Judith E. Unterlass is a scholar working on Molecular Biology, Biochemistry, Cancer Research, Genetics and Oncology, having authored 7 papers that have together received 121 indexed citations. Recurring topics across this work include Biochemical and Molecular Research (4 papers), Cancer, Hypoxia, and Metabolism (3 papers), Amino Acid Enzymes and Metabolism (2 papers), Metabolism and Genetic Disorders (1 paper), Cytomegalovirus and herpesvirus research (1 paper), Metabolism, Diabetes, and Cancer (1 paper), ATP Synthase and ATPases Research (1 paper) and Protein Kinase Regulation and GTPase Signaling (1 paper). The work is most often cited by research in Biochemistry (34 citations), Cancer Research (59 citations), Molecular Biology (93 citations), Clinical Biochemistry (8 citations) and Physiology (4 citations). Judith E. Unterlass has collaborated with scholars based in Sweden, United Kingdom and Belgium. Frequent co-authors include Nicola J. Curtin, M.E.M. Noble, Arnaud Baslé, Céline Cano, Julie A. Tucker, Timothy J. Blackburn, Séverine Ravez, Raphaël Frédérick, Quentin Spillier and Cyril Corbet. Their work appears in journals such as Oncotarget, MedChemComm, Pharmaceuticals, FEBS Journal and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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