Jonas Sundbäck

796 citations
17 papers · 686 · h-index 13

Impact in

  • Immunology top 5%
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Immunotherapy and Immune Responses
    • Reproductive System and Pregnancy
    • CAR-T cell therapy research

Papers in

    • Immune Cell Function and Interaction 17
    • T-cell and B-cell Immunology 13
    • Immunotherapy and Immune Responses 5
    • Toxin Mechanisms and Immunotoxins 1
    • HIV Research and Treatment 2

Jonas Sundbäck

17 papers receiving 672 citations

Peers

Jonas Sundbäck
Comparison fields: 5 of 42
  • Immunology 631
  • Oncology 114
  • Virology 17
  • Hematology 32
  • Epidemiology 43
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Citations per field
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Citations per year

Countries citing papers authored by Jonas Sundbäck

Since Specialization
Citations

This map shows the geographic impact of Jonas Sundbäck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonas Sundbäck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonas Sundbäck more than expected).

Fields of papers citing papers by Jonas Sundbäck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonas Sundbäck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonas Sundbäck. The network helps show where Jonas Sundbäck may publish in the future.

Co-authors

The 25 scholars most cited alongside Jonas Sundbäck, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonas Sundbäck Line = papers co-authored together Jonas Sundbäck links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 1997134
2 1997113
3 200068
4 200066
5 199863
6 199956
7 199633
8
The alpha2 domain of H-2Dd restricts the allelic specificity of the murine NK cell inhibitory receptor Ly-49A.
199831
9 199829
10 199925
11 199918
12 199818
13 200213
14 199711
15 20064
16 20003
17 19991

About Jonas Sundbäck

Jonas Sundbäck is a scholar working on Immunology, Virology, Oncology, Organic Chemistry and Molecular Biology, having authored 17 papers that have together received 686 indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (17 papers), T-cell and B-cell Immunology (13 papers), Immunotherapy and Immune Responses (5 papers), CAR-T cell therapy research (2 papers), HIV Research and Treatment (2 papers), Toxin Mechanisms and Immunotoxins (1 paper), Glycosylation and Glycoproteins Research (1 paper) and Carbohydrate Chemistry and Synthesis (1 paper). The work is most often cited by research in Immunology (631 citations), Oncology (114 citations), Virology (17 citations), Hematology (32 citations) and Epidemiology (43 citations). Jonas Sundbäck has collaborated with scholars based in Sweden, France and United States. Frequent co-authors include Klas Kärre, Margarita Salcedo, Adnane Achour, M Y Olsson-Alheim, Charles L. Sentman, Hans‐Gustaf Ljunggren, Robert Adam Harris, Luc Van Kaer, Alexander D. Diehl and Jakob Michaëlsson. Their work appears in journals such as The Journal of Immunology, European Journal of Immunology, International Journal of Cancer, Immunology Letters and Immunological Reviews.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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