Jon Weil

2.2k citations
59 papers · 1.9k · h-index 24

Impact in

  • Genetics top 2%
    • Bacterial Genetics and Biotechnology
    • BRCA gene mutations in cancer
  • Ecology top 5%
    • Bacteriophages and microbial interactions

Papers in

    • Bacterial Genetics and Biotechnology 12
    • BRCA gene mutations in cancer 11
    • RNA and protein synthesis mechanisms 6

Jon Weil

57 papers receiving 1.7k citations

Peers

Jon Weil
Comparison fields: 5 of 113
  • Genetics 841
  • Ecology 584
  • Pediatrics, Perinatology and Child Health 258
  • Molecular Biology 883
  • Immunology 189
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Citations per field
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Citations per year

Countries citing papers authored by Jon Weil

Since Specialization
Citations

This map shows the geographic impact of Jon Weil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon Weil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon Weil more than expected).

Fields of papers citing papers by Jon Weil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon Weil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon Weil. The network helps show where Jon Weil may publish in the future.

Co-authors

The 25 scholars most cited alongside Jon Weil, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jon Weil Line = papers co-authored together Jon Weil links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 59 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1968260
2 1983213
3 2000185
4 2003115
5 1968102
6 197592
7 197480
8 200652
9 196949
10 198046
11 200146
12 197243
13
The effect of trisomy 21 on the patterns of polypeptide synthesis in human fibroblasts.
197940
14 196837
15 201736
16 198135
17 196432
18 199331
19 198229
20 201828

About Jon Weil

Jon Weil is a scholar working on Genetics, Molecular Biology, Pediatrics, Perinatology and Child Health, Ecology and General Health Professions, having authored 59 papers that have together received 1.9k indexed citations. Recurring topics across this work include Bacteriophages and microbial interactions (14 papers), Bacterial Genetics and Biotechnology (12 papers), BRCA gene mutations in cancer (11 papers), Ethics and Legal Issues in Pediatric Healthcare (9 papers), Prenatal Screening and Diagnostics (8 papers), Counseling Practices and Supervision (6 papers), RNA and protein synthesis mechanisms (6 papers) and Family and Disability Support Research (6 papers). The work is most often cited by research in Genetics (841 citations), Ecology (584 citations), Pediatrics, Perinatology and Child Health (258 citations), Molecular Biology (883 citations) and Immunology (189 citations). Jon Weil has collaborated with scholars based in United States, Australia and Switzerland. Frequent co-authors include Ethan R. Signer, David Henderson, Lois B. Epstein, Charles J. Epstein, Theodore Homyk, J. J. Sedmak, Sidney E. Grossberg, J L Sabran, C. J. Epstein and David R. Cox. Their work appears in journals such as Journal of Molecular Biology, Virology, Genetics, Evolution and Journal of Genetic Counseling.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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