John G. Compton

9.3k citations

61 papers · 7.7k · 2 hit papers · h-index 34

Impact in

Dermatology top 0.05%
- Dermatology and Skin Diseases
- Contact Dermatitis and Allergies
Immunology and Allergy top 0.2%
- Allergic Rhinitis and Sensitization
- Food Allergy and Anaphylaxis Research

Papers in

Cell Biology 32
- Skin and Cellular Biology Research 28
- melanin and skin pigmentation 5
Urology 10
- Hair Growth and Disorders 10

Co-authors: Sherri J. Bale (30 shared papers)John J. DiGiovanna (14 shared papers)Peter M. Steinert (10 shared papers)Geraldine R. Rogers (5 shared papers)Bert W. O’Malley (7 shared papers)William T. Schrader (7 shared papers)Bernhard P. Korge (6 shared papers)Laura Russell (4 shared papers)
Journals: Journal of Investigative Dermatology (12 papers)Nature Genetics (9 papers)Genetics in Medicine (4 papers)Genomics (4 papers)Human Genetics (2 papers)
Partner nations: United States Italy United Kingdom

In The Last Decade

John G. Compton

60 papers receiving 7.3k citations

John G. Compton's Hit Papers

align trajectories log scale

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis 2006 · 2.4k citations

What are hit papers?

Paper score: A paper's citations measured against the top-1% bar of its own field and year: the square root of citations ÷ bar, so 1 means right at the bar and 2 means four times its citations. The bar blends the citation counts that enter the top 1% of the paper's subfields in its year (weighted 89.5%), of those subfields over all years (0.5%) and of its year across all fields (10%); a subfield and year with fewer than 400 papers takes its year's bar. Papers from 2026 are not scored yet: in an unfinished year, when a paper appeared matters more than how it is cited.

Hit paper: A paper cited at least 1.5× the top-1% bar of its field and year: a paper score of at least √1.5.

2006 Nature Genetics
2006 Nature Genetics

Peers

Countries citing papers authored by John G. Compton

Since Specialization

Citations

This map shows the geographic impact of John G. Compton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John G. Compton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John G. Compton more than expected).

Fields of papers citing papers by John G. Compton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John G. Compton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John G. Compton. The network helps show where John G. Compton may publish in the future.

Co-authors

The 25 scholars most cited alongside John G. Compton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John G. Compton Line = papers co-authored together John G. Compton links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 61 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis Nature Genetics ·Alan David Irvine, Ana Terron-Kwiatkowski, Yiwei Zhao, Haihui Liao, Simon P. Lee, David R. Goudie, Aileen Sandilands, Linda Elisabet Campbell Hit paper breakdown →	2006	2403
2	Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris Nature Genetics ·Frances J.D. Smith, Alan David Irvine, Ana Terron-Kwiatkowski, Aileen Sandilands, Linda Elisabet Campbell, Yiwei Zhao, Haihui Liao, Alan T Evans Hit paper breakdown →	2006	818
3	Assuring the quality of next-generation sequencing in clinical laboratory practice Nature Biotechnology ·Amy S. Gargis, Lisa V. Kalman, Meredith W Berry, David P Bick, David Dimmock, Tina M. Hambuch, Fei Lu, Elaine Lyon	2012	381
4	An evidence-based approach to establish the functional and clinical significance of copy number variants in intellectual and developmental disabilities Genetics in Medicine ·Erin B. Kaminsky, Vineith Kaul, Justin E. Paschall, Deanna M. Church, Brian P. Bunke, Dawn Kunig, Daniel Moreno‐De‐Luca, Andrés Moreno-De-Luca	2011	352
5	Mutations in the gene for transglutaminase 1 in autosomal recessive lamellar ichthyosis Nature Genetics ·Laura Russell, John J. DiGiovanna, Geraldine R. Rogers, Peter M. Steinert, Nemat Hashem, John G. Compton, Sherri J. Bale	1995	319
6	Mutations in the human connexin gene GJB3 cause erythrokeratodermia variabilis Nature Genetics ·Gabriele Richard, Lisa E. Smith, Regina A. Bailey, Peter H Itin, Daniel Hohl, Ervin H. Epstein, John J. DiGiovanna, John G. Compton	1998	291
7	A leucine→proline mutation in the H1 subdomain of keratin 1 causes epidermolytic hyperkeratosis Cell ·Constantin C. Chipev, Bernhard P. Korge, Nedialka G. Markova, Sherri J. Bale, John J. DiGiovanna, John G. Compton, Peter M. Steinert	1992	251
8	Functional defects of Cx26 resulting from a heterozygous missense mutation in a family with dominant deaf-mutism and palmoplantar keratoderma Human Genetics ·Gabriela Richard, Thomas W. White, Lisa E. Smith, Regina A. Bailey, John G. Compton, David L. Paul, Sherri J. Bale	1998	240
9	Sjögren–Larsson syndrome is caused by mutations in the fatty aldehyde dehydrogenase gene Nature Genetics ·Vincenzo De Laurenzi, Geraldine R. Rogers, David J. Hamrock, Lyuben N. Marekov, Peter M. Steinert, John G. Compton, Nelli Markova, William B. Rizzo	1996	220
10	Mutation of a new gene causes a unique form of Hermansky–Pudlak syndrome in a genetic isolate of central Puerto Rico Nature Genetics ·Yair Anikster, Marjan Huizing, James G. White, Yuriy O. Shevchenko, Diana L. Fitzpatrick, Jeffrey W. Touchman, John G. Compton, Sherri J. Bale	2001	186
11	DNA sequence preference of the progesterone receptor. Proceedings of the National Academy of Sciences ·John G. Compton, William T. Schrader, Bert W. O’Malley	1983	138
12	Physical Mapping of a Functional Cluster of Epidermal Differentiation Genes on Chromosome 1q21 Genomics ·Armin Volz, Bernhard P. Korge, John G. Compton, Andreas Ziegler, Peter M. Steinert, Dietmar Mischke	1993	134
13	Allosteric regulation of aspartate transcarbamoylase. Analysis of the structural and functional behavior in terms of a two-state model Biochemistry ·G.J. Howlett, Michael N. Blackburn, John G. Compton, H. K. Schachman	1977	128
14	A Mutation in the V1 End Domain of Keratin 1 in Non-Epidermolytic Palmar-Plantar Keratoderma Journal of Investigative Dermatology ·Virginia E. Kimonis, Jun-Mo Yang, Sharon Z Doyle, Sherri J. Bale, John G. Compton +1 other	1994	124
15	Genetic Heterogeneity in Erythrokeratodermia Variabilis: Novel Mutations in the Connexin Gene GJB4 (Cx30.3) and Genotype-Phenotype Correlations Journal of Investigative Dermatology ·Gabriele Richard, Nkecha Brown, Fatima Rouan, CARMEN D. CAMPANELLI, Jouni Uitto, Jan-Gerrit van der Schroeff, Emilia Bijlsma, Lawrence F. Eichenfield	2003	107
16	Prevalence and nonrandom distribution of exonic mutations in interferon regulatory factor 6 in 307 families with Van der Woude syndrome and 37 families with popliteal pterygium syndrome Genetics in Medicine ·Renata Lúcia Leite Ferreira de Lima, Sarah A. Hoper, Michella Ghassibe‐Sabbagh, Margaret E. Cooper, Nicholas K. Rorick, Shinji Kondo, Lori Katz, Mary L. Marazita	2009	107
17	A highly conserved lysine residue on the head domain of type II keratins is essential for the attachment of keratin intermediate filaments to the cornified cell envelope through isopeptide crosslinking by transglutaminases Proceedings of the National Academy of Sciences ·Eleonora Candi, Edit Tarcsa, John J. DiGiovanna, John G. Compton, Peter M. Elias, Lyuben N. Marekov, Peter M. Steinert	1998	102
18	Keratin 13 point mutation underlies the hereditary mucosal epithelia disorder white sponge nevus Nature Genetics ·Gabriela Richard, Vincenzo De Laurenzi, Biagio Didona, Sherri J. Bale, John G. Compton	1995	101
19	Linkage of epidermolytic hyperkeratosis to the type II keratin gene cluster on chromosome 12q Nature Genetics ·John G. Compton, John J. DiGiovanna, Sandra Santucci, Kathleen S. Kearns, Christopher I. Amos, Donita L. Abangan, Bernhard P. Korge, O. Wesley McBride	1992	89
20	Linkage of autosomal recessive lamellar ichthyosis to chromosome 14q. PubMed ·Laura Russell, John J. DiGiovanna, Nemat Hashem, John G. Compton, Sherri J. Bale	1994	83

About John G. Compton

John G. Compton is a scholar working on Cell Biology, Urology, Molecular Biology, Dermatology and Genetics, having authored 61 papers that have together received 7.7k indexed citations. Recurring topics across this work include Skin and Cellular Biology Research (28 papers), Hair Growth and Disorders (10 papers), Plant Reproductive Biology (7 papers), melanin and skin pigmentation (5 papers), Wnt/β-catenin signaling in development and cancer (4 papers), RNA Research and Splicing (4 papers), Hedgehog Signaling Pathway Studies (4 papers) and Biochemical and Molecular Research (3 papers). The work is most often cited by research in Dermatology (2.8k citations), Immunology and Allergy (1.5k citations), Cell Biology (2.3k citations), Urology (505 citations) and Sensory Systems (281 citations). John G. Compton has collaborated with scholars based in United States, Italy and United Kingdom. Frequent co-authors include Sherri J. Bale, John J. DiGiovanna, Peter M. Steinert, Geraldine R. Rogers, Bert W. O’Malley, William T. Schrader, Bernhard P. Korge, Laura Russell, Nemat Hashem and Lyuben N. Marekov. Their work appears in journals such as Journal of Investigative Dermatology, Nature Genetics, Genetics in Medicine, Genomics and Human Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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John G. Compton

John G. Compton's Hit Papers

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