John Down
Impact in
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- Nicotinic Acetylcholine Receptors Study
- Receptor Mechanisms and Signaling
- Ion channel regulation and function
- Chemical Synthesis and Analysis
Papers in
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- Nicotinic Acetylcholine Receptors Study 4
- Ion channel regulation and function 3
- Receptor Mechanisms and Signaling 2
- Chemical Synthesis and Analysis 1
- Co-authors
- Bruce G. Livett (4 shared papers)Zeinab G. Khalil (3 shared papers)Ken R. Gayler (3 shared papers)David Sandall (3 shared papers)David A. Keays (2 shared papers)Paul F. Alewood (1 shared paper)H.A. Robertson (1 shared paper)Peter Irwin (1 shared paper)
- Journals
- Journal of Parasitology (1 paper)European Journal of Pharmacology (1 paper)Brain Research (1 paper)Toxicon (1 paper)IEEE Engineering in Medicine and Biology Magazine (1 paper)
- Partner nations
- AustraliaUnited StatesNew Zealand
In The Last Decade
John Down
6 papers receiving 340 citations
Peers
Comparison fields: 5 of 61
- Molecular Biology 291
- Parasitology 21
- Cellular and Molecular Neuroscience 57
- Microbiology 18
- Pharmacology 39
Countries citing papers authored by John Down
This map shows the geographic impact of John Down's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Down with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Down more than expected).
Fields of papers citing papers by John Down
This network shows the impact of papers produced by John Down. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Down. The network helps show where John Down may publish in the future.
Co-authors
The 16 scholars most cited alongside John Down, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2005 | 167 | |
| 2 | 2006 | 115 | |
| 3 | 2008 | 22 | |
| 4 | 2007 | 19 | |
| 5 | 2000 | 17 | |
| 6 | 2005 | 13 |
About John Down
John Down is a scholar working on Molecular Biology, Control and Systems Engineering, Ecology, Evolution, Behavior and Systematics, Physiology and Microbiology, having authored 6 papers that have together received 353 indexed citations. Recurring topics across this work include Nicotinic Acetylcholine Receptors Study (4 papers), Ion channel regulation and function (3 papers), Receptor Mechanisms and Signaling (2 papers), Advanced Sensor Technologies Research (1 paper), Bird parasitology and diseases (1 paper), Vector-borne infectious diseases (1 paper), Pain Mechanisms and Treatments (1 paper) and Chemical Synthesis and Analysis (1 paper). The work is most often cited by research in Molecular Biology (291 citations), Parasitology (21 citations), Cellular and Molecular Neuroscience (57 citations), Microbiology (18 citations) and Pharmacology (39 citations). John Down has collaborated with scholars based in Australia, United States and New Zealand. Frequent co-authors include Bruce G. Livett, Zeinab G. Khalil, Ken R. Gayler, David Sandall, David A. Keays, Paul F. Alewood, H.A. Robertson, Peter Irwin, Linda M. McInnes and Una Ryan. Their work appears in journals such as Journal of Parasitology, European Journal of Pharmacology, Brain Research, Toxicon and IEEE Engineering in Medicine and Biology Magazine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.