John C. Law

858 citations
16 papers · 659 · h-index 12

Impact in

Papers in

    • Cancer-related gene regulation 4
    • RNA modifications and cancer 3
    • Epigenetics and DNA Methylation 2
    • Peroxisome Proliferator-Activated Receptors 1
    • DNA Repair Mechanisms 1
    • Cancer-related Molecular Pathways 7

John C. Law

16 papers receiving 643 citations

Peers

John C. Law
Comparison fields: 5 of 73
  • Otorhinolaryngology 46
  • Oncology 275
  • Cancer Research 116
  • Molecular Biology 321
  • Hematology 40
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C Kittas Greece
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Citations per field
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Citations per year

Countries citing papers authored by John C. Law

Since Specialization
Citations

This map shows the geographic impact of John C. Law's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John C. Law with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John C. Law more than expected).

Fields of papers citing papers by John C. Law

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John C. Law. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John C. Law. The network helps show where John C. Law may publish in the future.

Co-authors

The 25 scholars most cited alongside John C. Law, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John C. Law Line = papers co-authored together John C. Law links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 1993132
2 2006125
3
A germ line mutation in exon 5 of the p53 gene in an extended cancer family.
199176
4 199767
5 198555
6 199547
7
Assessment of sensitivity and specificity of immunohistochemical staining of p53 in lung and head and neck cancers.
199547
8 199641
9 199818
10 199516
11 199214
12 199611
13 19964
14 19922
15
Deletion and mutational inactivation of the cyclin-dependent kinase-4 inhibitor gene (p16) in oral carcinoma
19942
16 19882

About John C. Law

John C. Law is a scholar working on Molecular Biology, Oncology, Surgery, Cancer Research and Otorhinolaryngology, having authored 16 papers that have together received 659 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (7 papers), Cancer-related gene regulation (4 papers), Cancer Genomics and Diagnostics (3 papers), RNA modifications and cancer (3 papers), Epigenetics and DNA Methylation (2 papers), Cholangiocarcinoma and Gallbladder Cancer Studies (2 papers), Peroxisome Proliferator-Activated Receptors (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Otorhinolaryngology (46 citations), Oncology (275 citations), Cancer Research (116 citations), Molecular Biology (321 citations) and Hematology (40 citations). John C. Law has collaborated with scholars based in United States, Poland and United Kingdom. Frequent co-authors include Robert E. Ferrell, Jack C. Yalowich, Gerd Leder, Jonas T. Johnson, Eugene N. Myers, Chandramohan S. Ishwad, Susanne M. Gollin, Abirami Chidambaram, Louise C. Strong and Kenneth L. Garver. Their work appears in journals such as International Journal of Cancer, Analytical Biochemistry, Clinical Genetics, Genomics and Lung Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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