John A. Conrad

526 citations
25 papers · 409 · h-index 13

Impact in

Papers in

John A. Conrad

24 papers receiving 401 citations

Peers

John A. Conrad
Comparison fields: 5 of 81
  • Developmental Neuroscience 21
  • Neurology 61
  • Inorganic Chemistry 50
  • Molecular Biology 241
  • Cellular and Molecular Neuroscience 60
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Citations per year

Countries citing papers authored by John A. Conrad

Since Specialization
Citations

This map shows the geographic impact of John A. Conrad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John A. Conrad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John A. Conrad more than expected).

Fields of papers citing papers by John A. Conrad

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John A. Conrad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John A. Conrad. The network helps show where John A. Conrad may publish in the future.

Co-authors

The 25 scholars most cited alongside John A. Conrad, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John A. Conrad Line = papers co-authored together John A. Conrad links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 25 papers — load more, or switch the sort, to bring in the rest.

#Work
1 200974
2 199935
3 201132
4 201130
5 199530
6 199629
7 201227
8 199618
9 201017
10 201316
11 202115
12 201415
13 199514
14 201711
15 20149
16 19779
17 20078
18 19967
19
Functional effects of transplanted human fetal ventral mesencephalic brain tissue from spontaneous abortions into a rodent model of Parkinson's disease.
19943
20
[Hemorrhagic syndrome and isolated alpha 2-antiplasmin deficiency. Apropos of a case].
19972

About John A. Conrad

John A. Conrad is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Neurology, Inorganic Chemistry and Developmental Neuroscience, having authored 25 papers that have together received 409 indexed citations. Recurring topics across this work include Metal-Catalyzed Oxygenation Mechanisms (4 papers), Biochemical and Molecular Research (3 papers), Nerve injury and regeneration (2 papers), Innovative Teaching Methods (2 papers), Pharmacogenetics and Drug Metabolism (2 papers), Neurological disorders and treatments (2 papers), Genetic Neurodegenerative Diseases (2 papers) and Microbial metabolism and enzyme function (2 papers). The work is most often cited by research in Developmental Neuroscience (21 citations), Neurology (61 citations), Inorganic Chemistry (50 citations), Molecular Biology (241 citations) and Cellular and Molecular Neuroscience (60 citations). John A. Conrad has collaborated with scholars based in United States, Norway and Tunisia. Frequent co-authors include Walter C. Low, Graham R. Moran, Takeshi Kondoh, Bruce A. Palfey, Scott A. Lesley, Amnon Kohen, Irimpan I. Mathews, Daniel Wahl, Patrick O’Brien and Jason Chesney. Their work appears in journals such as Biochemistry, Cell Transplantation, Journal of the American Chemical Society, Neuroscience Research and Neurosurgery.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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