James Velier

881 citations
8 papers · 737 · h-index 7

Impact in

    • Genetic Neurodegenerative Diseases
    • Neuroscience and Neuropharmacology Research
  • Neurology top 10%
    • Neuroinflammation and Neurodegeneration Mechanisms
    • Neurological disorders and treatments

Papers in

James Velier

8 papers receiving 726 citations

Peers

James Velier
Comparison fields: 5 of 67
  • Cellular and Molecular Neuroscience 379
  • Neurology 111
  • Developmental Neuroscience 45
  • Neurology 121
  • Molecular Biology 493
Replace Laura Davis Keppen with:
Laura Davis Keppen United States
Bala T. S. Susarla United States
Mitsuhiro Otani Japan
Jennifer A. Cameron United States
Annelies Quaegebeur United Kingdom
Yuen Ling Lee United States
Bärbel Schäfer Germany
Sònia Sirisi Spain
Óscar G. Vidaurre Spain
Masakiyo Sasahara Japan
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Citations per field
00.5×2.6×
Laura Davis Keppen · 1×
Citations per year

Countries citing papers authored by James Velier

Since Specialization
Citations

This map shows the geographic impact of James Velier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James Velier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James Velier more than expected).

Fields of papers citing papers by James Velier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James Velier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James Velier. The network helps show where James Velier may publish in the future.

Co-authors

The 25 scholars most cited alongside James Velier, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with James Velier Line = papers co-authored together James Velier links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 1998247
2 1999237
3 1998148
4 199945
5 199939
6 199713
7 19996
8 20252

About James Velier

James Velier is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Neurology, Rheumatology and Neurology, having authored 8 papers that have together received 737 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (4 papers), Mitochondrial Function and Pathology (3 papers), Neurological disorders and treatments (2 papers), Neuroscience and Neuropharmacology Research (2 papers), Muscle Physiology and Disorders (2 papers), Hippo pathway signaling and YAP/TAZ (1 paper), Neurogenesis and neuroplasticity mechanisms (1 paper) and Trace Elements in Health (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (379 citations), Neurology (111 citations), Developmental Neuroscience (45 citations), Neurology (121 citations) and Molecular Biology (493 citations). James Velier has collaborated with scholars based in United States, Canada and United Kingdom. Frequent co-authors include Julie A. Ellison, Frank C. Barone, Giora Feuerstein, Kristine Kikly, Neil Aronin, Ellen Sapp, Kathryn Chase, Marian DiFiglia, Manho Kim and Tae Wan Kim. Their work appears in journals such as The Journal of Comparative Neurology, Stroke, Neuroscience, Journal of Biological Chemistry and Experimental Neurology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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