James E. Haley

532 citations
11 papers · 443 · h-index 9

Impact in

Papers in

James E. Haley

11 papers receiving 414 citations

Peers

James E. Haley
Comparison fields: 5 of 65
  • Developmental Neuroscience 55
  • Cellular and Molecular Neuroscience 146
  • Clinical Biochemistry 37
  • Neurology 44
  • Cell Biology 67
Replace H P Siegrist with:
H P Siegrist Switzerland
M. G. Tavaststjerna United States
K. H. Chou United States
Naoki Hamajima Japan
J.W. Crabb United States
Simon Ngamli Fewou Germany
Un‐Jin P. Zimmerman United States
Afshin Yaghootfam Germany
Clive Palfrey United States
Moulinath Acharya India
James E. Haley relative to H P Siegrist Switzerland H P Siegrist's profile →
Citations per field
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H P Siegrist · 1×
Citations per year

Countries citing papers authored by James E. Haley

Since Specialization
Citations

This map shows the geographic impact of James E. Haley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Haley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Haley more than expected).

Fields of papers citing papers by James E. Haley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Haley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Haley. The network helps show where James E. Haley may publish in the future.

Co-authors

The 10 scholars most cited alongside James E. Haley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with James E. Haley Line = papers co-authored together James E. Haley links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 196595
2 197572
3 198167
4 197946
5 198144
6 197935
7 197928
8 198327
9 199216
10 19837
11 19746

About James E. Haley

James E. Haley is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Developmental Neuroscience, Neurology and Neurology, having authored 11 papers that have together received 443 indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (3 papers), Neuroscience and Neuropharmacology Research (3 papers), Lipid Membrane Structure and Behavior (2 papers), Cellular transport and secretion (2 papers), Neurological Disorders and Treatments (2 papers), Glycosylation and Glycoproteins Research (1 paper), Ion Transport and Channel Regulation (1 paper) and Fatty Acid Research and Health (1 paper). The work is most often cited by research in Developmental Neuroscience (55 citations), Cellular and Molecular Neuroscience (146 citations), Clinical Biochemistry (37 citations), Neurology (44 citations) and Cell Biology (67 citations). James E. Haley has collaborated with scholars based in United States. Frequent co-authors include Robert W. Ledeen, George Brecher, Stephen H. Robinson, James A. Miller, Susan C. Specht, Bernice Grafstein, Henryk M. Wı́sniewski, H Kjeldbye, Peter Gouras and M T Flood. Their work appears in journals such as Journal of Neurochemistry, Brain Research, Analytical Biochemistry, Molecular Neurobiology and Cellular and Molecular Neurobiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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