J. A. Eisman

12 papers receiving 802 citations

Peers

J. A. Eisman
Comparison fields: 5 of 80
  • Orthopedics and Sports Medicine 250
  • Pathology and Forensic Medicine 506
  • Genetics 230
  • Endocrinology, Diabetes and Metabolism 108
  • Nutrition and Dietetics 93
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Citations per year

Countries citing papers authored by J. A. Eisman

Since Specialization
Citations

This map shows the geographic impact of J. A. Eisman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. A. Eisman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. A. Eisman more than expected).

Fields of papers citing papers by J. A. Eisman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. A. Eisman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. A. Eisman. The network helps show where J. A. Eisman may publish in the future.

Co-authors

The 25 scholars most cited alongside J. A. Eisman, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with J. A. Eisman Line = papers co-authored together J. A. Eisman links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 1992480
2
Presence of 1,25-dihydroxyvitamin D3 receptors in established human cancer cell lines in culture.
1982115
3
Regulation of epidermal growth factor receptor levels by 1,25-dihydroxyvitamin D3 in human breast cancer cells.
198882
4 197646
5
Frequency of 1,25-dihydroxyvitamin D3 receptor in human breast cancer.
198134
6 197424
7 198120
8
Pharmacogenomics of osteoporosis: opportunities and challenges.
200619
9 198112
10 19974
11 19854
12
Calcitonin and 1,25-dihydroxycholecalciferol receptors in human breast cancer cells
19802

About J. A. Eisman

J. A. Eisman is a scholar working on Pathology and Forensic Medicine, Genetics, Endocrinology, Diabetes and Metabolism, Molecular Biology and Surgery, having authored 12 papers that have together received 842 indexed citations. Recurring topics across this work include Vitamin D Research Studies (7 papers), Estrogen and related hormone effects (5 papers), Growth Hormone and Insulin-like Growth Factors (3 papers), Bone Metabolism and Diseases (2 papers), Cancer, Stress, Anesthesia, and Immune Response (2 papers), Pancreatic function and diabetes (1 paper), Bone health and osteoporosis research (1 paper) and Effects and risks of endocrine disrupting chemicals (1 paper). The work is most often cited by research in Orthopedics and Sports Medicine (250 citations), Pathology and Forensic Medicine (506 citations), Genetics (230 citations), Endocrinology, Diabetes and Metabolism (108 citations) and Nutrition and Dietetics (93 citations). J. A. Eisman has collaborated with scholars based in Australia and Switzerland. Frequent co-authors include Paul J. Kelly, N A Morrison, T. John Martin, R. Sutherland, Masafumi Koga, Larry J. Suva, J. M. Moseley, David M. Findlay, Ruth Frampton and G. E. Moore. Their work appears in journals such as Journal of Endocrinology, Clinical Science, Hormone and Metabolic Research, Proceedings of the National Academy of Sciences and World review of nutrition and dietetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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