Ingrid Gemperlein
Impact in
- Hematology top 10%
- Acute Myeloid Leukemia Research
- Chronic Myeloid Leukemia Treatments
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- Carcinogens and Genotoxicity Assessment
Papers in
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- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2
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- Chemical Reactions and Isotopes 2
- Co-authors
- Hansruedi Glatt (3 shared papers)Franz Oesch (3 shared papers)M Valtieri (2 shared papers)D Santoli (2 shared papers)Fulvio Mavilio (2 shared papers)Daniele Caracciolo (2 shared papers)Beverly J. Lange (2 shared papers)J Finan (1 shared paper)
- Journals
- Nucleic Acids Research (2 papers)Mutagenesis (2 papers)The Journal of Immunology (1 paper)Blood (1 paper)PubMed (1 paper)
- Partner nations
- United StatesGermany
In The Last Decade
Ingrid Gemperlein
8 papers receiving 470 citations
Peers
Comparison fields: 5 of 61
- Hematology 138
- Cancer Research 120
- Immunology 128
- Pharmacology 47
- Molecular Biology 214
Countries citing papers authored by Ingrid Gemperlein
This map shows the geographic impact of Ingrid Gemperlein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingrid Gemperlein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingrid Gemperlein more than expected).
Fields of papers citing papers by Ingrid Gemperlein
This network shows the impact of papers produced by Ingrid Gemperlein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingrid Gemperlein. The network helps show where Ingrid Gemperlein may publish in the future.
Co-authors
The 25 scholars most cited alongside Ingrid Gemperlein, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1987 | 200 | |
| 2 | 1990 | 91 | |
| 3 | Search for cell culture systems with diverse xenobiotic-metabolizing activities and their use in toxicological studies. | 1989 | 58 |
| 4 | 1987 | 43 | |
| 5 | 1989 | 35 | |
| 6 | 1989 | 32 | |
| 7 | 1987 | 18 | |
| 8 | 1997 | 5 |
About Ingrid Gemperlein
Ingrid Gemperlein is a scholar working on Immunology, Pharmaceutical Science, Surgery, Molecular Biology and Ecology, Evolution, Behavior and Systematics, having authored 8 papers that have together received 482 indexed citations. Recurring topics across this work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Chemical Reactions and Isotopes (2 papers), Acute Myeloid Leukemia Research (1 paper), Estrogen and related hormone effects (1 paper), Endometriosis Research and Treatment (1 paper), Acute Lymphoblastic Leukemia research (1 paper), Telomeres, Telomerase, and Senescence (1 paper) and Amino Acid Enzymes and Metabolism (1 paper). The work is most often cited by research in Hematology (138 citations), Cancer Research (120 citations), Immunology (128 citations), Pharmacology (47 citations) and Molecular Biology (214 citations). Ingrid Gemperlein has collaborated with scholars based in United States and Germany. Frequent co-authors include Hansruedi Glatt, Franz Oesch, M Valtieri, D Santoli, Fulvio Mavilio, Daniele Caracciolo, Beverly J. Lange, J Finan, Beverly S. Emanuel and C. A. Griffin. Their work appears in journals such as Nucleic Acids Research, Mutagenesis, The Journal of Immunology, Blood and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.