I-Lin Ho
Impact in
- General Materials Science top 10%
Papers in
-
- Ubiquitin and proteasome pathways 3
- Cell death mechanisms and regulation 2
- DNA Repair Mechanisms 1
- Protein Degradation and Inhibitors 1
- Oncology 6
- Cancer-related Molecular Pathways 3
- Peptidase Inhibition and Analysis 2
- Co-authors
- I. Egry (1 shared paper)Hong‐Chiang Chang (5 shared papers)Kuo‐How Huang (5 shared papers)Wei‐Chou Lin (5 shared papers)Kuan-Lin Kuo (5 shared papers)Yeong‐Shiau Pu (5 shared papers)Ju‐Ton Hsieh (4 shared papers)Chung‐Sheng Shi (4 shared papers)
- Journals
- PLoS ONE (2 papers)Nature Communications (1 paper)Cancer Letters (1 paper)Drug and Alcohol Dependence (1 paper)Toxicological Sciences (1 paper)
- Partner nations
- TaiwanUnited StatesGermany
In The Last Decade
I-Lin Ho
11 papers receiving 294 citations
Peers
Comparison fields: 5 of 71
- General Materials Science 13
- Aging 6
- Oncology 76
- Molecular Biology 169
- Cancer Research 27
Countries citing papers authored by I-Lin Ho
This map shows the geographic impact of I-Lin Ho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I-Lin Ho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I-Lin Ho more than expected).
Fields of papers citing papers by I-Lin Ho
This network shows the impact of papers produced by I-Lin Ho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I-Lin Ho. The network helps show where I-Lin Ho may publish in the future.
Co-authors
The 25 scholars most cited alongside I-Lin Ho, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 54 | |
| 2 | 2006 | 54 | |
| 3 | 2015 | 40 | |
| 4 | 2019 | 34 | |
| 5 | 2013 | 25 | |
| 6 | MLN4924, a Novel NEDD8-activating enzyme inhibitor, exhibits antitumor activity and enhances cisplatin-induced cytotoxicity in human cervical carcinoma: in vitro and in vivo study. | 2015 | 22 |
| 7 | 2021 | 21 | |
| 8 | 2013 | 20 | |
| 9 | 2018 | 16 | |
| 10 | 1999 | 7 | |
| 11 | 2014 | 1 |
About I-Lin Ho
I-Lin Ho is a scholar working on Molecular Biology, Oncology, Epidemiology, Health, Toxicology and Mutagenesis and Physiology, having authored 11 papers that have together received 294 indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (3 papers), Cancer-related Molecular Pathways (3 papers), Peptidase Inhibition and Analysis (2 papers), Cell death mechanisms and regulation (2 papers), DNA Repair Mechanisms (1 paper), Protein Degradation and Inhibitors (1 paper), Toxin Mechanisms and Immunotoxins (1 paper) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in General Materials Science (13 citations), Aging (6 citations), Oncology (76 citations), Molecular Biology (169 citations) and Cancer Research (27 citations). I-Lin Ho has collaborated with scholars based in Taiwan, United States and Germany. Frequent co-authors include I. Egry, Hong‐Chiang Chang, Kuo‐How Huang, Wei‐Chou Lin, Kuan-Lin Kuo, Yeong‐Shiau Pu, Ju‐Ton Hsieh, Chung‐Sheng Shi, June‐Tai Wu and Yu‐Chieh Tsai. Their work appears in journals such as PLoS ONE, Nature Communications, Cancer Letters, Drug and Alcohol Dependence and Toxicological Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.