I. Ott

663 citations
13 papers · 535 · h-index 6

Impact in

Papers in

I. Ott

12 papers receiving 522 citations

Peers

I. Ott
Comparison fields: 5 of 68
  • Internal Medicine 91
  • Hematology 199
  • Cardiology and Cardiovascular Medicine 268
  • Immunology and Allergy 65
  • Biochemistry 27
Replace G. Pellegrini with:
G. Pellegrini Italy
Kenzo Sugi Japan
David Gurney United Kingdom
H. M. Sanderson United Kingdom
Corinna Böttiger Germany
Melanie McCabe White United States
Miry Blich Israel
Dominik Weiss Germany
Kousuke Kumeda Japan
John C. Kovacich United States
I. Ott relative to G. Pellegrini Italy G. Pellegrini's profile →
Citations per field
00.5×1.7×
G. Pellegrini · 1×
Citations per year

Countries citing papers authored by I. Ott

Since Specialization
Citations

This map shows the geographic impact of I. Ott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Ott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Ott more than expected).

Fields of papers citing papers by I. Ott

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by I. Ott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Ott. The network helps show where I. Ott may publish in the future.

Co-authors

The 19 scholars most cited alongside I. Ott, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with I. Ott Line = papers co-authored together I. Ott links everyone, so they are left out of the graph.

All Works

13 of 13 papers shown
#Work
1 1996327
2
Effects of magnesium on platelet aggregation and adhesion. Magnesium modulates surface expression of glycoproteins on platelets in vitro and ex vivo.
1994107
3 199738
4 200835
5 199311
6
Neutrophil hyper-reactivity after exercise-induced angina pectoris.
19955
7 20084
8 20103
9 20092
10 20081
11 20081
12 20091
13 20230

About I. Ott

I. Ott is a scholar working on Surgery, Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine, Pathology and Forensic Medicine and Rheumatology, having authored 13 papers that have together received 535 indexed citations. Recurring topics across this work include Antiplatelet Therapy and Cardiovascular Diseases (4 papers), Platelet Disorders and Treatments (2 papers), Acute Myocardial Infarction Research (2 papers), Head and Neck Surgical Oncology (2 papers), Soft tissue tumor case studies (1 paper), Urticaria and Related Conditions (1 paper), Microbial bioremediation and biosurfactants (1 paper) and Nasal Surgery and Airway Studies (1 paper). The work is most often cited by research in Internal Medicine (91 citations), Hematology (199 citations), Cardiology and Cardiovascular Medicine (268 citations), Immunology and Allergy (65 citations) and Biochemistry (27 citations). I. Ott has collaborated with scholars based in Germany and Nigeria. Frequent co-authors include Meinrad Gawaz, Franz‐Josef Neumann, Manfred Schmitt, Albert Schömig, Andreas E. May, Hanna Walter, S Braun, Dirk Sibbing, G Busch and N. von Beckerath. Their work appears in journals such as European Heart Journal, Heart, Circulation, HNO and Laryngo-Rhino-Otologie.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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