Hye-Won Hyun
Impact in
- Neurology top 10%
- Neuroinflammation and Neurodegeneration Mechanisms
- Barrier Structure and Function Studies
- Physiology top 10%
- Adenosine and Purinergic Signaling
- Alzheimer's disease research and treatments
Papers in
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- Mitochondrial Function and Pathology 2
-
- Neuroscience and Neuropharmacology Research 5
- Co-authors
- Ji‐Eun Kim (5 shared papers)Tae‐Cheon Kang (8 shared papers)Ji‐Eun Kim (3 shared papers)Jieun Kim (1 shared paper)Duk-Shin Lee (2 shared papers)Min Ju Kim (1 shared paper)Yeon-Joo Kim (1 shared paper)
- Journals
- Frontiers in Cellular Neuroscience (3 papers)Neuroscience (2 papers)Brain Research (2 papers)Scientific Reports (2 papers)Frontiers in Molecular Neuroscience (1 paper)
- Partner nations
- South Korea
In The Last Decade
Hye-Won Hyun
13 papers receiving 341 citations
Peers
Comparison fields: 5 of 59
- Neurology 70
- Physiology 33
- Cellular and Molecular Neuroscience 88
- Cell Biology 71
- Clinical Biochemistry 24
Countries citing papers authored by Hye-Won Hyun
This map shows the geographic impact of Hye-Won Hyun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hye-Won Hyun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hye-Won Hyun more than expected).
Fields of papers citing papers by Hye-Won Hyun
This network shows the impact of papers produced by Hye-Won Hyun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hye-Won Hyun. The network helps show where Hye-Won Hyun may publish in the future.
Co-authors
The 7 scholars most cited alongside Hye-Won Hyun, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 67 | |
| 2 | 2015 | 48 | |
| 3 | 2018 | 41 | |
| 4 | 2017 | 34 | |
| 5 | 2016 | 22 | |
| 6 | 2017 | 20 | |
| 7 | 2015 | 20 | |
| 8 | 2015 | 20 | |
| 9 | 2016 | 19 | |
| 10 | 2017 | 18 | |
| 11 | 2015 | 15 | |
| 12 | 2017 | 9 | |
| 13 | 2017 | 9 |
About Hye-Won Hyun
Hye-Won Hyun is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Cell Biology, Neurology and Epidemiology, having authored 13 papers that have together received 342 indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (5 papers), Neuroscience and Neuropharmacology Research (5 papers), Adenosine and Purinergic Signaling (2 papers), Mitochondrial Function and Pathology (2 papers), Autophagy in Disease and Therapy (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Genetics and Neurodevelopmental Disorders (1 paper) and Neurogenesis and neuroplasticity mechanisms (1 paper). The work is most often cited by research in Neurology (70 citations), Physiology (33 citations), Cellular and Molecular Neuroscience (88 citations), Cell Biology (71 citations) and Clinical Biochemistry (24 citations). Hye-Won Hyun has collaborated with scholars based in South Korea. Frequent co-authors include Ji‐Eun Kim, Tae‐Cheon Kang, Ji‐Eun Kim, Jieun Kim, Duk-Shin Lee, Min Ju Kim and Yeon-Joo Kim. Their work appears in journals such as Frontiers in Cellular Neuroscience, Neuroscience, Brain Research, Scientific Reports and Frontiers in Molecular Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.