Hiroka Iida

661 citations
6 papers · 565 · h-index 5

Impact in

  • Cell Biology top 10%
    • Endoplasmic Reticulum Stress and Disease
    • Microtubule and mitosis dynamics
    • DNA Repair Mechanisms
    • Ubiquitin and proteasome pathways
    • Nuclear Structure and Function
    • RNA Research and Splicing
    • Genomics and Chromatin Dynamics

Papers in

Hiroka Iida

6 papers receiving 564 citations

Peers

Hiroka Iida
Comparison fields: 5 of 66
  • Cell Biology 208
  • Molecular Biology 467
  • Physiology 28
  • Oncology 132
  • Aging 7
Replace T. Kotenyova with:
T. Kotenyova Sweden
Petra Mikolčević United Kingdom
Norbert Volkmar United Kingdom
Gabriela Ondrovičová Slovakia
John Fielden United Kingdom
Alejandro Carpy Germany
Scott Rusin United States
Beata Kuśmider Switzerland
Fred Wittinghofer Germany
Tara L. Muratore‐Schroeder United States
Hiroka Iida relative to T. Kotenyova Sweden T. Kotenyova's profile →
Citations per field
00.5×3.1×
T. Kotenyova · 1×
Citations per year

Countries citing papers authored by Hiroka Iida

Since Specialization
Citations

This map shows the geographic impact of Hiroka Iida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroka Iida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroka Iida more than expected).

Fields of papers citing papers by Hiroka Iida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroka Iida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroka Iida. The network helps show where Hiroka Iida may publish in the future.

Co-authors

The 25 scholars most cited alongside Hiroka Iida, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hiroka Iida Line = papers co-authored together Hiroka Iida links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 2008201
2 1991168
3 2010110
4 200967
5 200615
6 20004

About Hiroka Iida

Hiroka Iida is a scholar working on Molecular Biology, Cell Biology, Aquatic Science, Oceanography and Ocean Engineering, having authored 6 papers that have together received 565 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (2 papers), Plant Molecular Biology Research (1 paper), Galectins and Cancer Biology (1 paper), Cancer-related Molecular Pathways (1 paper), Bacterial Genetics and Biotechnology (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper), Machine Learning in Materials Science (1 paper) and Echinoderm biology and ecology (1 paper). The work is most often cited by research in Cell Biology (208 citations), Molecular Biology (467 citations), Physiology (28 citations), Oncology (132 citations) and Aging (7 citations). Hiroka Iida has collaborated with scholars based in Japan, Italy and Canada. Frequent co-authors include Hideo Nishitani, Yasushi Shiomi, Toshihiro Takami, Toshiki Tsurimoto, Kenji Inaba, Mamoru Suzuki, Motoaki Ohtsubo, Hiroki Yasudo, Tony Hunter and Tetsuya NISHIMOTO. Their work appears in journals such as The EMBO Journal, Reproduction, Journal of Biological Chemistry and Gene Expression Patterns.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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