Herbert Eradat

56 papers receiving 979 citations

Peers

Herbert Eradat
Comparison fields: 5 of 59
  • Genetics 556
  • Pathology and Forensic Medicine 472
  • Hematology 161
  • Immunology 236
  • Oncology 243
Replace Jerzy Z. Błoński with:
Jerzy Z. Błoński Poland
Alicja Gruszka Italy
Joanna Góra‐Tybor Poland
Sílvia Marcé Spain
Beth Christian United States
Amit Khot Australia
Mark Bishton United Kingdom
S. Öhl Germany
Dariusz Wołowiec Poland
Michael Voralia Canada
Herbert Eradat relative to Jerzy Z. Błoński Poland Jerzy Z. Błoński's profile →
Citations per field
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Jerzy Z. Błoński · 1×
Citations per year

Countries citing papers authored by Herbert Eradat

Since Specialization
Citations

This map shows the geographic impact of Herbert Eradat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Herbert Eradat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Herbert Eradat more than expected).

Fields of papers citing papers by Herbert Eradat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Herbert Eradat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Herbert Eradat. The network helps show where Herbert Eradat may publish in the future.

Co-authors

The 25 scholars most cited alongside Herbert Eradat, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Herbert Eradat Line = papers co-authored together Herbert Eradat links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 62 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2015154
2 1997114
3 201886
4 201557
5 202043
6 201641
7 202232
8 201630
9 201929
10 202228
11 202228
12 201120
13 201318
14 202118
15 201618
16 201317
17 201617
18 202315
19 202115
20 201815

About Herbert Eradat

Herbert Eradat is a scholar working on Genetics, Pathology and Forensic Medicine, Oncology, Pulmonary and Respiratory Medicine and Immunology, having authored 62 papers that have together received 990 indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (39 papers), Lymphoma Diagnosis and Treatment (33 papers), Advanced Breast Cancer Therapies (11 papers), CAR-T cell therapy research (11 papers), Immunodeficiency and Autoimmune Disorders (8 papers), Chronic Myeloid Leukemia Treatments (7 papers), Monoclonal and Polyclonal Antibodies Research (4 papers) and Cutaneous lymphoproliferative disorders research (4 papers). The work is most often cited by research in Genetics (556 citations), Pathology and Forensic Medicine (472 citations), Hematology (161 citations), Immunology (236 citations) and Oncology (243 citations). Herbert Eradat has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Thomas J. Kipps, Richard R. Furman, Ernest Brahn, Eli E. Sercarz, Tammy T. Chang, Radhey S. Gupta, Kamal D. Moudgil, Iryna Dyagil, Halyna Pylypenko and Deborah Hurst. Their work appears in journals such as Blood, Journal of Clinical Oncology, Hematological Oncology, The Lancet Haematology and HemaSphere.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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