H. Klinger
Impact in
- Aging top 5%
- Genetics, Aging, and Longevity in Model Organisms
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- Fungal and yeast genetics research
- Mitochondrial Function and Pathology
Papers in
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- Mitochondrial Function and Pathology 3
- Fungal and yeast genetics research 3
- Chemical Synthesis and Analysis 1
- CRISPR and Genetic Engineering 1
- Aging 2
- Genetics, Aging, and Longevity in Model Organisms 2
- Co-authors
- Mark Rinnerthaler (4 shared papers)Peter Laun (3 shared papers)Gino Heeren (4 shared papers)Michael Breitenbach (3 shared papers)Stefanie Jarolim (3 shared papers)Charlotte Delay (1 shared paper)Frank Madeo (3 shared papers)Klaartje Pellens (1 shared paper)
In The Last Decade
H. Klinger
6 papers receiving 305 citations
Peers
Comparison fields: 5 of 61
- Aging 73
- Molecular Biology 244
- Cellular and Molecular Neuroscience 63
- Cell Biology 42
- Geriatrics and Gerontology 9
Countries citing papers authored by H. Klinger
This map shows the geographic impact of H. Klinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Klinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Klinger more than expected).
Fields of papers citing papers by H. Klinger
This network shows the impact of papers produced by H. Klinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Klinger. The network helps show where H. Klinger may publish in the future.
Co-authors
The 25 scholars most cited alongside H. Klinger, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 90 | |
| 2 | 2008 | 84 | |
| 3 | 2009 | 70 | |
| 4 | 2010 | 60 | |
| 5 | 1967 | 7 | |
| 6 | The mitochondrial ribosomal of the large subunit, afo1p, determines cellular longevity through mitochondrial back-signaling via TOR1 | 2009 | 1 |
About H. Klinger
H. Klinger is a scholar working on Molecular Biology, Aging, Cellular and Molecular Neuroscience, Neurology and Pharmaceutical Science, having authored 6 papers that have together received 312 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (3 papers), Fungal and yeast genetics research (3 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers), Genetic Neurodegenerative Diseases (1 paper), Parkinson's Disease Mechanisms and Treatments (1 paper), Fluorine in Organic Chemistry (1 paper), Chemical Synthesis and Analysis (1 paper) and CRISPR and Genetic Engineering (1 paper). The work is most often cited by research in Aging (73 citations), Molecular Biology (244 citations), Cellular and Molecular Neuroscience (63 citations), Cell Biology (42 citations) and Geriatrics and Gerontology (9 citations). H. Klinger has collaborated with scholars based in Austria, France and Australia. Frequent co-authors include Mark Rinnerthaler, Peter Laun, Gino Heeren, Michael Breitenbach, Stefanie Jarolim, Charlotte Delay, Frank Madeo, Klaartje Pellens, Thomas Vanhelmont and Piotr Zabrocki. Their work appears in journals such as Journal of Medicinal Chemistry, Aging, Biochimica et Biophysica Acta (BBA) - Bioenergetics, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research and Experimental Gerontology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.