Guylhène Bourdat-Michel
Impact in
- Nephrology top 5%
- Renal Diseases and Glomerulopathies
- Chronic Kidney Disease and Diabetes
- Endocrinology top 10%
- Escherichia coli research studies
Papers in
-
- Renal Diseases and Glomerulopathies 2
- Genetics 2
- Connective tissue disorders research 1
- Co-authors
- Bruno Ranchin (2 shared papers)Pierre Cochat (2 shared papers)A. Liutkus (1 shared paper)François Nobili (1 shared paper)Jean‐Bernard Palcoux (1 shared paper)Marie‐Pierre Lavocat (1 shared paper)Rita Van Damme‐Lombaerts (1 shared paper)Lucie Bessenay (1 shared paper)
- Journals
- Pediatric Nephrology (3 papers)Clinical Journal of the American Society of Nephrology (1 paper)Archives de Pédiatrie (3 papers)
- Partner nations
- FranceSwitzerlandUnited States
In The Last Decade
Guylhène Bourdat-Michel
4 papers receiving 308 citations
Peers
Comparison fields: 5 of 52
- Nephrology 158
- Endocrinology 61
- Immunology 115
- Genetics 47
- Hematology 44
Countries citing papers authored by Guylhène Bourdat-Michel
This map shows the geographic impact of Guylhène Bourdat-Michel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guylhène Bourdat-Michel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guylhène Bourdat-Michel more than expected).
Fields of papers citing papers by Guylhène Bourdat-Michel
This network shows the impact of papers produced by Guylhène Bourdat-Michel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guylhène Bourdat-Michel. The network helps show where Guylhène Bourdat-Michel may publish in the future.
Co-authors
The 25 scholars most cited alongside Guylhène Bourdat-Michel, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 156 | |
| 2 | 2009 | 142 | |
| 3 | 1998 | 15 | |
| 4 | 2010 | 3 | |
| 5 | 2024 | 0 | |
| 6 | 1999 | 0 | |
| 7 | 2015 | 0 |
About Guylhène Bourdat-Michel
Guylhène Bourdat-Michel is a scholar working on Nephrology, Genetics, Genetics, Surgery and Molecular Biology, having authored 7 papers that have together received 316 indexed citations. Recurring topics across this work include Renal Diseases and Glomerulopathies (2 papers), Intestinal Malrotation and Obstruction Disorders (1 paper), Connective tissue disorders research (1 paper), Lysosomal Storage Disorders Research (1 paper), Nephrotoxicity and Medicinal Plants (1 paper), Complement system in diseases (1 paper), Iron Metabolism and Disorders (1 paper) and Congenital gastrointestinal and neural anomalies (1 paper). The work is most often cited by research in Nephrology (158 citations), Endocrinology (61 citations), Immunology (115 citations), Genetics (47 citations) and Hematology (44 citations). Guylhène Bourdat-Michel has collaborated with scholars based in France, Switzerland and United States. Frequent co-authors include Bruno Ranchin, Pierre Cochat, A. Liutkus, François Nobili, Jean‐Bernard Palcoux, Marie‐Pierre Lavocat, Rita Van Damme‐Lombaerts, Lucie Bessenay, Djalila Mekahli and François Cachat. Their work appears in journals such as Pediatric Nephrology, Clinical Journal of the American Society of Nephrology and Archives de Pédiatrie.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.