Guus Hateboer
Impact in
- Oncology top 5%
- Cancer-related Molecular Pathways
- Molecular Biology top 10%
- Ubiquitin and proteasome pathways
- Epigenetics and DNA Methylation
- RNA modifications and cancer
- DNA Repair Mechanisms
- Genomics and Chromatin Dynamics
Papers in
- Co-authors
- Rene Bernards (4 shared papers)Kristian Helin (3 shared papers)Elena Vigo (3 shared papers)Merlijn Bazuine (1 shared paper)Wilma T. Steegenga (1 shared paper)A.J. van der Eb (1 shared paper)Nicole Riteco (1 shared paper)Theo van Laar (1 shared paper)
- Journals
- Journal of Biological Chemistry (2 papers)Molecular and Cellular Biology (2 papers)The EMBO Journal (2 papers)Plant Science (1 paper)Biotechnology and Bioengineering (1 paper)
- Partner nations
- NetherlandsItalyFrance
In The Last Decade
Guus Hateboer
11 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 74
- Oncology 835
- Molecular Biology 1.3k
- Biotechnology 139
- Cancer Research 154
- Cell Biology 172
Countries citing papers authored by Guus Hateboer
This map shows the geographic impact of Guus Hateboer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guus Hateboer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guus Hateboer more than expected).
Fields of papers citing papers by Guus Hateboer
This network shows the impact of papers produced by Guus Hateboer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guus Hateboer. The network helps show where Guus Hateboer may publish in the future.
Co-authors
The 25 scholars most cited alongside Guus Hateboer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1996 | 534 | |
| 2 | 1999 | 284 | |
| 3 | 1998 | 161 | |
| 4 | 1993 | 134 | |
| 5 | 1991 | 124 | |
| 6 | 1997 | 115 | |
| 7 | 1995 | 76 | |
| 8 | 1996 | 66 | |
| 9 | 2007 | 34 | |
| 10 | 1992 | 12 | |
| 11 | Cell cycle regulated expression of mammalian CDC6 is dependent on E2F | 1998 | 11 |
About Guus Hateboer
Guus Hateboer is a scholar working on Oncology, Genetics, Molecular Biology, Immunology and Virology, having authored 11 papers that have together received 1.6k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (5 papers), Epigenetics and DNA Methylation (5 papers), Virus-based gene therapy research (4 papers), RNA modifications and cancer (2 papers), Viral Infectious Diseases and Gene Expression in Insects (2 papers), Ubiquitin and proteasome pathways (2 papers), Poxvirus research and outbreaks (1 paper) and Cancer Research and Treatments (1 paper). The work is most often cited by research in Oncology (835 citations), Molecular Biology (1.3k citations), Biotechnology (139 citations), Cancer Research (154 citations) and Cell Biology (172 citations). Guus Hateboer has collaborated with scholars based in Netherlands, Italy and France. Frequent co-authors include Rene Bernards, Kristian Helin, Elena Vigo, Merlijn Bazuine, Wilma T. Steegenga, A.J. van der Eb, Nicole Riteco, Theo van Laar, Анна Шварц and Aart G. Jochemsen. Their work appears in journals such as Journal of Biological Chemistry, Molecular and Cellular Biology, The EMBO Journal, Plant Science and Biotechnology and Bioengineering.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.