Gudrun Aspöck
Impact in
- Aging top 1%
- Genetics, Aging, and Longevity in Model Organisms
- Endocrine and Autonomic Systems top 10%
- Circadian rhythm and melatonin
Papers in
- Aging 7
- Genetics, Aging, and Longevity in Model Organisms 7
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- Epigenetics and DNA Methylation 2
- Pluripotent Stem Cells Research 2
- CRISPR and Genetic Engineering 2
- Hedgehog Signaling Pathway Studies 1
- Co-authors
- Thomas R. Bürglin (7 shared papers)Hiroshi Kagoshima (2 shared papers)Gisela Niklaus (2 shared papers)Rebecca D. Burdine (1 shared paper)Jens Erik Dietrich (1 shared paper)Monte Westerfield (1 shared paper)Sunit Dutta (1 shared paper)Zoltán Varga (1 shared paper)
- Journals
- Development (2 papers)Mechanisms of Development (1 paper)Developmental Biology (1 paper)Gene (1 paper)Development Genes and Evolution (1 paper)
- Partner nations
- SwitzerlandUnited StatesSweden
In The Last Decade
Gudrun Aspöck
8 papers receiving 400 citations
Peers
Comparison fields: 5 of 56
- Aging 206
- Endocrine and Autonomic Systems 66
- Molecular Biology 266
- Cell Biology 43
- Genetics 67
Countries citing papers authored by Gudrun Aspöck
This map shows the geographic impact of Gudrun Aspöck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gudrun Aspöck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gudrun Aspöck more than expected).
Fields of papers citing papers by Gudrun Aspöck
This network shows the impact of papers produced by Gudrun Aspöck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gudrun Aspöck. The network helps show where Gudrun Aspöck may publish in the future.
Co-authors
The 20 scholars most cited alongside Gudrun Aspöck, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1999 | 113 | |
| 2 | 2005 | 108 | |
| 3 | 2002 | 76 | |
| 4 | 2003 | 31 | |
| 5 | 1998 | 30 | |
| 6 | 2006 | 25 | |
| 7 | 2001 | 18 | |
| 8 | 1999 | 6 |
About Gudrun Aspöck
Gudrun Aspöck is a scholar working on Aging, Molecular Biology, Genetics, Cellular and Molecular Neuroscience and Endocrine and Autonomic Systems, having authored 8 papers that have together received 407 indexed citations. Recurring topics across this work include Genetics, Aging, and Longevity in Model Organisms (7 papers), Epigenetics and DNA Methylation (2 papers), Pluripotent Stem Cells Research (2 papers), CRISPR and Genetic Engineering (2 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (1 paper), Circadian rhythm and melatonin (1 paper), Digestive system and related health (1 paper) and Hedgehog Signaling Pathway Studies (1 paper). The work is most often cited by research in Aging (206 citations), Endocrine and Autonomic Systems (66 citations), Molecular Biology (266 citations), Cell Biology (43 citations) and Genetics (67 citations). Gudrun Aspöck has collaborated with scholars based in Switzerland, United States and Sweden. Frequent co-authors include Thomas R. Bürglin, Hiroshi Kagoshima, Gisela Niklaus, Rebecca D. Burdine, Jens Erik Dietrich, Monte Westerfield, Sunit Dutta, Zoltán Varga, Alexander F. Schier and Gary Ruvkun. Their work appears in journals such as Development, Mechanisms of Development, Developmental Biology, Gene and Development Genes and Evolution.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.