Gauri C. Bedi

2.0k citations
8 papers · 1.7k · h-index 8

Impact in

    • NF-κB Signaling Pathways
    • Cancer Genomics and Diagnostics
  • Oncology top 5%
    • Cancer-related Molecular Pathways

Papers in

    • Cancer-related Molecular Pathways 4
    • Viral-associated cancers and disorders 1
    • Cell death mechanisms and regulation 2
    • DNA Repair Mechanisms 1

Gauri C. Bedi

8 papers receiving 1.6k citations

Peers

Gauri C. Bedi
Comparison fields: 5 of 82
  • Cancer Research 436
  • Oncology 777
  • Otorhinolaryngology 99
  • Pathology and Forensic Medicine 256
  • Molecular Biology 864
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Citations per year

Countries citing papers authored by Gauri C. Bedi

Since Specialization
Citations

This map shows the geographic impact of Gauri C. Bedi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gauri C. Bedi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gauri C. Bedi more than expected).

Fields of papers citing papers by Gauri C. Bedi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gauri C. Bedi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gauri C. Bedi. The network helps show where Gauri C. Bedi may publish in the future.

Co-authors

The 25 scholars most cited alongside Gauri C. Bedi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gauri C. Bedi Line = papers co-authored together Gauri C. Bedi links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1
Inhibition of apoptosis during development of colorectal cancer.
1995485
2
A novel p16INK4A transcript.
1995282
3 2001268
4
Multiple head and neck tumors: evidence for a common clonal origin.
1996221
5 1996139
6
p53-mediated repression of nuclear factor-kappaB RelA via the transcriptional integrator p300.
1998139
7 199575
8
AIDS-related Kaposi's sarcoma is a clonal neoplasm.
199551

About Gauri C. Bedi

Gauri C. Bedi is a scholar working on Oncology, Molecular Biology, Pathology and Forensic Medicine, Immunology and Cancer Research, having authored 8 papers that have together received 1.7k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (4 papers), Genetic factors in colorectal cancer (3 papers), interferon and immune responses (2 papers), NF-κB Signaling Pathways (2 papers), Lymphoma Diagnosis and Treatment (2 papers), Cell death mechanisms and regulation (2 papers), DNA Repair Mechanisms (1 paper) and Viral-associated cancers and disorders (1 paper). The work is most often cited by research in Cancer Research (436 citations), Oncology (777 citations), Otorhinolaryngology (99 citations), Pathology and Forensic Medicine (256 citations) and Molecular Biology (864 citations). Gauri C. Bedi has collaborated with scholars based in United States. Frequent co-authors include David Sidransky, A Bedi, Adil Akhtar, Rajani Ravi, Ephraim J. Fuchs, Pankaj J. Pasricha, Barbara A. Zehnbauer, Stanley R. Hamilton, Richard J. Jones and Francis M. Giardiello. Their work appears in journals such as Nature Medicine, Nature Cell Biology and PubMed.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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