Étienne Hébert Chatelain

451 citations
8 papers · 369 · h-index 6

Impact in

  • Aging top 10%
    • Genetics, Aging, and Longevity in Model Organisms
    • Autophagy in Disease and Therapy

Papers in

    • Mitochondrial Function and Pathology 5
    • ATP Synthase and ATPases Research 2
    • Protein Tyrosine Phosphatases 1
    • Physiological and biochemical adaptations 2
    • Crustacean biology and ecology 1

Étienne Hébert Chatelain

8 papers receiving 367 citations

Peers

Étienne Hébert Chatelain
Comparison fields: 5 of 73
  • Aging 24
  • Epidemiology 142
  • Geriatrics and Gerontology 16
  • Molecular Biology 235
  • Physiology 14
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Nicolas Offner France
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Citations per field
00.5×4.7×
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Citations per year

Countries citing papers authored by Étienne Hébert Chatelain

Since Specialization
Citations

This map shows the geographic impact of Étienne Hébert Chatelain's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Étienne Hébert Chatelain with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Étienne Hébert Chatelain more than expected).

Fields of papers citing papers by Étienne Hébert Chatelain

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Étienne Hébert Chatelain. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Étienne Hébert Chatelain. The network helps show where Étienne Hébert Chatelain may publish in the future.

Co-authors

The 23 scholars most cited alongside Étienne Hébert Chatelain, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Étienne Hébert Chatelain Line = papers co-authored together Étienne Hébert Chatelain links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2013216
2 201060
3 201032
4 201121
5 200718
6 201812
7 20105
8 20085

About Étienne Hébert Chatelain

Étienne Hébert Chatelain is a scholar working on Molecular Biology, Ecology, Pharmaceutical Science, Oceanography and Geriatrics and Gerontology, having authored 8 papers that have together received 369 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (5 papers), ATP Synthase and ATPases Research (2 papers), Physiological and biochemical adaptations (2 papers), Aquatic Ecosystems and Phytoplankton Dynamics (1 paper), Evolution and Genetic Dynamics (1 paper), Protein Tyrosine Phosphatases (1 paper), Crustacean biology and ecology (1 paper) and Chemical Reactions and Isotopes (1 paper). The work is most often cited by research in Aging (24 citations), Epidemiology (142 citations), Geriatrics and Gerontology (16 citations), Molecular Biology (235 citations) and Physiology (14 citations). Étienne Hébert Chatelain has collaborated with scholars based in Canada, France and Australia. Frequent co-authors include Caroline José, Giovanni Bénard, Rodrigue Rossignol, Susan Goorden, Émilie Obre, Julie Lavie, Walid Mahfouf, Ype Elgersma, Hamid Rezvani and Muriel Priault. Their work appears in journals such as Cell Metabolism, Journal of Experimental Biology, Journal of Thermal Biology, Mitochondrion and Cellular and Molecular Life Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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