Ekaterina Gresko

774 citations
16 papers · 614 · h-index 11

Impact in

  • Oncology top 10%
    • Cancer-related Molecular Pathways
    • Cancer Cells and Metastasis
    • Cancer Risks and Factors
  • Cell Biology top 10%
    • Hippo pathway signaling and YAP/TAZ
    • Microtubule and mitosis dynamics

Papers in

    • Ubiquitin and proteasome pathways 3
    • Cell death mechanisms and regulation 3
    • Retinoids in leukemia and cellular processes 2
    • Cancer-related Molecular Pathways 6

Ekaterina Gresko

14 papers receiving 600 citations

Peers

Ekaterina Gresko
Comparison fields: 5 of 57
  • Oncology 256
  • Cell Biology 143
  • Molecular Biology 400
  • Cancer Research 74
  • Genetics 38
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Citations per field
00.5×1.5×2.4×
Matthew D. Smith · 1×
Citations per year

Countries citing papers authored by Ekaterina Gresko

Since Specialization
Citations

This map shows the geographic impact of Ekaterina Gresko's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ekaterina Gresko with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ekaterina Gresko more than expected).

Fields of papers citing papers by Ekaterina Gresko

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ekaterina Gresko. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ekaterina Gresko. The network helps show where Ekaterina Gresko may publish in the future.

Co-authors

The 25 scholars most cited alongside Ekaterina Gresko, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ekaterina Gresko Line = papers co-authored together Ekaterina Gresko links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 2008134
2 2010106
3 200698
4 200661
5 201253
6 200539
7 200335
8 200833
9 201820
10 201813
11 200412
12 20196
13 20253
14 20231
15 20260
16 20230

About Ekaterina Gresko

Ekaterina Gresko is a scholar working on Molecular Biology, Oncology, Hematology, Immunology and Genetics, having authored 16 papers that have together received 614 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (6 papers), Ubiquitin and proteasome pathways (3 papers), Lymphoma Diagnosis and Treatment (3 papers), Immunodeficiency and Autoimmune Disorders (3 papers), Cell death mechanisms and regulation (3 papers), Chronic Lymphocytic Leukemia Research (3 papers), Hemophilia Treatment and Research (2 papers) and Retinoids in leukemia and cellular processes (2 papers). The work is most often cited by research in Oncology (256 citations), Cell Biology (143 citations), Molecular Biology (400 citations), Cancer Research (74 citations) and Genetics (38 citations). Ekaterina Gresko has collaborated with scholars based in Switzerland, Germany and Italy. Frequent co-authors include Anton Vichalkovski, Brian A. Hemmings, M. Lienhard Schmitz, Ana Roscic, Andreas Möller, Hauke Cornils, Alexander Hergovich, Debora Schmitz, David F. Restuccia and Marco A. Calzado. Their work appears in journals such as Oncogene, Leukemia, Molecular Cell, Biochemical and Biophysical Research Communications and Current Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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