E.H. Sage

492 citations
7 papers · 433 · h-index 6

Impact in

Papers in

    • Bone and Dental Protein Studies 4
    • Folate and B Vitamins Research 1
    • Connective tissue disorders research 2

E.H. Sage

7 papers receiving 410 citations

Peers

E.H. Sage
Comparison fields: 5 of 71
  • Immunology and Allergy 97
  • Hematology 158
  • Nephrology 67
  • Internal Medicine 13
  • Cancer Research 47
Replace Petra Vandervoort with:
Petra Vandervoort Belgium
Nagaharu Tsukiji Japan
JR Maynard United States
V. S. Rukosuev Russia
Jane Hudson United States
Luigi De Marco Italy
Katrina Ashworth United States
Sarah Eichenberger United States
Gunilla Grundström Sweden
E.H. Sage relative to Petra Vandervoort Belgium Petra Vandervoort's profile →
Citations per field
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Petra Vandervoort · 1×
Citations per year

Countries citing papers authored by E.H. Sage

Since Specialization
Citations

This map shows the geographic impact of E.H. Sage's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E.H. Sage with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E.H. Sage more than expected).

Fields of papers citing papers by E.H. Sage

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E.H. Sage. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E.H. Sage. The network helps show where E.H. Sage may publish in the future.

Co-authors

The 22 scholars most cited alongside E.H. Sage, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E.H. Sage Line = papers co-authored together E.H. Sage links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 1994212
2
Markers of complement-dependent and complement-independent glomerular visceral epithelial cell injury in vivo. Expression of antiadhesive proteins and cytoskeletal changes.
1992101
3 197747
4 199132
5 199726
6 199410
7 19995

About E.H. Sage

E.H. Sage is a scholar working on Rheumatology, Genetics, Immunology and Allergy, Molecular Biology and Pulmonary and Respiratory Medicine, having authored 7 papers that have together received 433 indexed citations. Recurring topics across this work include Bone and Dental Protein Studies (4 papers), Cell Adhesion Molecules Research (2 papers), Connective tissue disorders research (2 papers), Folate and B Vitamins Research (1 paper), Williams Syndrome Research (1 paper), Blood properties and coagulation (1 paper), Infant Nutrition and Health (1 paper) and Mercury impact and mitigation studies (1 paper). The work is most often cited by research in Immunology and Allergy (97 citations), Hematology (158 citations), Nephrology (67 citations), Internal Medicine (13 citations) and Cancer Research (47 citations). E.H. Sage has collaborated with scholars based in United States and Austria. Frequent co-authors include William R. Gray, H. R. Gralnick, J J Sixma, Laurie P. McKeown, Sybil B. Williams, PG de Groot, H K Nieuwenhuis, Charles E. Alpers, Richard J. Johnson and William G. Couser. Their work appears in journals such as British Journal of Cancer, Nature Biotechnology, Advances in experimental medicine and biology, Blood and Archives of Biochemistry and Biophysics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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