E.A. Siess

2.3k citations
60 papers · 2.1k · h-index 23

Impact in

Papers in

E.A. Siess

57 papers receiving 1.9k citations

Peers

E.A. Siess
Comparison fields: 5 of 94
  • Clinical Biochemistry 740
  • Biochemistry 648
  • Physiology 702
  • Cell Biology 304
  • Molecular Biology 1.2k
Replace F.A. Hommes with:
F.A. Hommes Netherlands
Luisa Raijman United States
Paul A. Weinhold United States
Darold Holten United States
C.R. Park United States
Gary W. Goodwin United States
Rob Ofman Netherlands
Annemarie Herzfeld United States
G. Dacremont Belgium
Stephanie J. Mihalik United States
E.A. Siess relative to F.A. Hommes Netherlands F.A. Hommes's profile →
Citations per field
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F.A. Hommes · 1×
Citations per year

Countries citing papers authored by E.A. Siess

Since Specialization
Citations

This map shows the geographic impact of E.A. Siess's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E.A. Siess with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E.A. Siess more than expected).

Fields of papers citing papers by E.A. Siess

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E.A. Siess. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E.A. Siess. The network helps show where E.A. Siess may publish in the future.

Co-authors

The 25 scholars most cited alongside E.A. Siess, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E.A. Siess Line = papers co-authored together E.A. Siess links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 60 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1977227
2 1971214
3 1980143
4 1976129
5 1970127
6 1971122
7 1978115
8 1972103
9 197688
10 197554
11 198148
12 197845
13 197245
14 198044
15 198544
16 198243
17 198037
18 198335
19 197834
20 197830

About E.A. Siess

E.A. Siess is a scholar working on Molecular Biology, Clinical Biochemistry, Biochemistry, Physiology and Surgery, having authored 60 papers that have together received 2.1k indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (21 papers), Biochemical Acid Research Studies (15 papers), Diet and metabolism studies (12 papers), Amino Acid Enzymes and Metabolism (10 papers), Pancreatic function and diabetes (9 papers), Mitochondrial Function and Pathology (9 papers), Metabolism, Diabetes, and Cancer (8 papers) and Liver Disease Diagnosis and Treatment (8 papers). The work is most often cited by research in Clinical Biochemistry (740 citations), Biochemistry (648 citations), Physiology (702 citations), Cell Biology (304 citations) and Molecular Biology (1.2k citations). E.A. Siess has collaborated with scholars based in Germany, China and United States. Frequent co-authors include O. Wieland, D G Brocks, Josef Wittmann, R. Dolhofer, Felix Wieland, Ursula Hartmann, Monika Hofstetter, Feodor Lynen, Thomas Schreckenbach and Jeffrey L. Fox. Their work appears in journals such as FEBS Letters, European Journal of Biochemistry, Diabetologia, Biochemical Journal and Analytical Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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