E. Tulchinsky

497 citations
8 papers · 426 · h-index 6

Impact in

Papers in

    • Cancer-related gene regulation 3
    • RNA modifications and cancer 3
    • RNA and protein synthesis mechanisms 2
    • DNA Repair Mechanisms 1
    • RNA Research and Splicing 1
    • Animal Genetics and Reproduction 1

E. Tulchinsky

8 papers receiving 422 citations

Peers

E. Tulchinsky
Comparison fields: 5 of 50
  • Cancer Research 127
  • Immunology and Allergy 43
  • Molecular Biology 378
  • Immunology 64
  • Oncology 48
Replace M Kaufmann with:
M Kaufmann Germany
Laia Cubells Spain
Kenneth van Golen United States
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Lufeng Liang China
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Sébastien Vallée United States
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Citations per field
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Citations per year

Countries citing papers authored by E. Tulchinsky

Since Specialization
Citations

This map shows the geographic impact of E. Tulchinsky's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Tulchinsky with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Tulchinsky more than expected).

Fields of papers citing papers by E. Tulchinsky

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Tulchinsky. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Tulchinsky. The network helps show where E. Tulchinsky may publish in the future.

Co-authors

The 17 scholars most cited alongside E. Tulchinsky, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E. Tulchinsky Line = papers co-authored together E. Tulchinsky links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 1989259
2 199377
3 198529
4
Characterization of a positive regulatory element in the mts1 gene.
199327
5
Novel AP-1 binding site created by DNA-methylation.
199625
6 19907
7
Characterization of a positive regulatory element in the mts1 gene
19921
8
Structural characterization of mts271 - a new gene for calcium-binding protein
19891

About E. Tulchinsky

E. Tulchinsky is a scholar working on Molecular Biology, Genetics, Immunology, Cancer Research and Infectious Diseases, having authored 8 papers that have together received 426 indexed citations. Recurring topics across this work include Cancer-related gene regulation (3 papers), RNA modifications and cancer (3 papers), RNA and protein synthesis mechanisms (2 papers), interferon and immune responses (1 paper), DNA Repair Mechanisms (1 paper), NF-κB Signaling Pathways (1 paper), Animal Genetics and Reproduction (1 paper) and RNA Research and Splicing (1 paper). The work is most often cited by research in Cancer Research (127 citations), Immunology and Allergy (43 citations), Molecular Biology (378 citations), Immunology (64 citations) and Oncology (48 citations). E. Tulchinsky has collaborated with scholars based in Russia, United States and Denmark. Frequent co-authors include Mariam Grigorian, Eugene Lukanidin, Alexander K. Ebralidze, Е. С. Ревазова, Arina Afanasyeva, E. M. Lukanidin, Marina Kriajevska, Sayeeda B. Zain, Georgii P. Georgiev and Dmitri A. Kramerov. Their work appears in journals such as Gene, The EMBO Journal, Genes & Development, FEBS Letters and Genetika.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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