E Satoyoshi
Impact in
- Neurology top 1%
- Peripheral Neuropathies and Disorders
- Myasthenia Gravis and Thymoma
-
- Nerve injury and regeneration
- Genetic Neurodegenerative Diseases
- Hereditary Neurological Disorders
Papers in
-
- Muscle Physiology and Disorders 12
- Mitochondrial Function and Pathology 7
- Ion channel regulation and function 6
- Neurology 34
- Myasthenia Gravis and Thymoma 16
- Autoimmune Neurological Disorders and Treatments 7
- Co-authors
- N Sunohara (23 shared papers)Yoshiko Furukawa (9 shared papers)Ikuya Nonaka (11 shared papers)Shoei Furukawa (13 shared papers)Kyozo Hayashi (8 shared papers)Masao Kinoshita (18 shared papers)Shoichi Ishiura (1 shared paper)I Sobue (2 shared papers)
- Journals
- Neurology (9 papers)Journal of the Neurological Sciences (7 papers)Annals of Neurology (4 papers)Biochemical and Biophysical Research Communications (4 papers)Journal of Neurology (3 papers)
- Partner nations
- JapanUnited StatesSouth Africa
In The Last Decade
E Satoyoshi
99 papers receiving 2.6k citations
Peers
Comparison fields: 5 of 105
- Neurology 798
- Cellular and Molecular Neuroscience 929
- Developmental Neuroscience 148
- Genetics 208
- Rheumatology 251
Countries citing papers authored by E Satoyoshi
This map shows the geographic impact of E Satoyoshi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E Satoyoshi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E Satoyoshi more than expected).
Fields of papers citing papers by E Satoyoshi
This network shows the impact of papers produced by E Satoyoshi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E Satoyoshi. The network helps show where E Satoyoshi may publish in the future.
Co-authors
The 25 scholars most cited alongside E Satoyoshi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 111 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 1984 | 424 | |
| 2 | 1981 | 234 | |
| 3 | 1987 | 110 | |
| 4 | 1983 | 107 | |
| 5 | 1987 | 105 | |
| 6 | 1989 | 91 | |
| 7 | 1985 | 85 | |
| 8 | 1994 | 83 | |
| 9 | 1977 | 82 | |
| 10 | 1986 | 68 | |
| 11 | 1989 | 63 | |
| 12 | 1963 | 61 | |
| 13 | 1986 | 58 | |
| 14 | 1985 | 58 | |
| 15 | 1982 | 56 | |
| 16 | 1963 | 54 | |
| 17 | 1984 | 54 | |
| 18 | 1978 | 53 | |
| 19 | 1986 | 47 | |
| 20 | 1984 | 43 |
About E Satoyoshi
E Satoyoshi is a scholar working on Molecular Biology, Neurology, Cellular and Molecular Neuroscience, Physiology and Rheumatology, having authored 111 papers that have together received 2.8k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (19 papers), Myasthenia Gravis and Thymoma (16 papers), Muscle Physiology and Disorders (12 papers), Nerve injury and regeneration (8 papers), Metabolism and Genetic Disorders (8 papers), Autoimmune Neurological Disorders and Treatments (7 papers), Mitochondrial Function and Pathology (7 papers) and Ion channel regulation and function (6 papers). The work is most often cited by research in Neurology (798 citations), Cellular and Molecular Neuroscience (929 citations), Developmental Neuroscience (148 citations), Genetics (208 citations) and Rheumatology (251 citations). E Satoyoshi has collaborated with scholars based in Japan, United States and South Africa. Frequent co-authors include N Sunohara, Yoshiko Furukawa, Ikuya Nonaka, Shoei Furukawa, Kyozo Hayashi, Masao Kinoshita, Shoichi Ishiura, I Sobue, Y Toyokura and T Tsubaki. Their work appears in journals such as Neurology, Journal of the Neurological Sciences, Annals of Neurology, Biochemical and Biophysical Research Communications and Journal of Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.