E. Mathias

2.1k citations
11 papers · 298 · h-index 6

Impact in

Papers in

    • HER2/EGFR in Cancer Research 4
    • Cancer Treatment and Pharmacology 3
    • Autoimmune Neurological Disorders and Treatments 3

E. Mathias

9 papers receiving 294 citations

Peers

E. Mathias
Comparison fields: 5 of 46
  • Neurology 168
  • Biological Psychiatry 19
  • Oncology 84
  • Genetics 70
  • Cellular and Molecular Neuroscience 43
Replace Chloé Bost with:
Chloé Bost France
Aurélien Viaccoz France
Krystyna M. Jones United States
Luis Dávila-Maldonado Mexico
J.‐C. Antoine France
Martin Köhnlein Germany
James Varley United Kingdom
Esther Hulsenboom Netherlands
Nese Sinmaz Australia
Anastasie Dunn‐Pirio United States
E. Mathias relative to Chloé Bost France Chloé Bost's profile →
Citations per field
00.5×3.8×
Chloé Bost · 1×
Citations per year

Countries citing papers authored by E. Mathias

Since Specialization
Citations

This map shows the geographic impact of E. Mathias's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Mathias with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Mathias more than expected).

Fields of papers citing papers by E. Mathias

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Mathias. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Mathias. The network helps show where E. Mathias may publish in the future.

Co-authors

The 25 scholars most cited alongside E. Mathias, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E. Mathias Line = papers co-authored together E. Mathias links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 201796
2 201595
3 202350
4 202324
5 201316
6 201213
7 20242
8 20251
9 20211
10 20240
11 20150

About E. Mathias

E. Mathias is a scholar working on Oncology, Neurology, Pulmonary and Respiratory Medicine, Epidemiology and Radiology, Nuclear Medicine and Imaging, having authored 11 papers that have together received 298 indexed citations. Recurring topics across this work include HER2/EGFR in Cancer Research (4 papers), Cancer Treatment and Pharmacology (3 papers), Autoimmune Neurological Disorders and Treatments (3 papers), Cytomegalovirus and herpesvirus research (2 papers), Breast Cancer Treatment Studies (2 papers), Genetics and Neurodevelopmental Disorders (1 paper), Hematological disorders and diagnostics (1 paper) and Thermal Regulation in Medicine (1 paper). The work is most often cited by research in Neurology (168 citations), Biological Psychiatry (19 citations), Oncology (84 citations), Genetics (70 citations) and Cellular and Molecular Neuroscience (43 citations). E. Mathias has collaborated with scholars based in France, United States and Japan. Frequent co-authors include Jérôme Honnorat, Véronique Rogemond, François Ducray, Kumaran Deiva, Αναστασία Ζεκερίδου, Frédéric Villega, Cyril Gitiaux, Jean‐Yves Delattre, Marc Tardieu and Aurélien Viaccoz. Their work appears in journals such as Annals of Oncology, Nature Communications, ESMO Open, Diagnostic and Interventional Imaging and Journal of Neurology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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