E Jacotot
Impact in
- Virology top 10%
- HIV Research and Treatment
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- Metabolism and Genetic Disorders
Papers in
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- Mitochondrial Function and Pathology 2
- Cell death mechanisms and regulation 1
- Retinoids in leukemia and cellular processes 1
- ATP Synthase and ATPases Research 1
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- HIV/AIDS drug development and treatment 1
- Co-authors
- Guido Kroemer (4 shared papers)Catherine Brenner (3 shared papers)Helena L.A. Vieira (2 shared papers)D. Haouzi (1 shared paper)Chahrazed El Hamel (1 shared paper)Nathanaël Larochette (1 shared paper)Zhihua Xie (1 shared paper)L Ravagnan (1 shared paper)
- Journals
- APOPTOSIS (1 paper)Cell Death and Disease (1 paper)Cell Death and Differentiation (1 paper)PubMed (2 papers)
- Partner nations
- FranceUnited Kingdom
In The Last Decade
E Jacotot
5 papers receiving 405 citations
Peers
Comparison fields: 5 of 78
- Virology 46
- Clinical Biochemistry 32
- Molecular Biology 290
- Aging 6
- Cancer Research 37
Countries citing papers authored by E Jacotot
This map shows the geographic impact of E Jacotot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E Jacotot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E Jacotot more than expected).
Fields of papers citing papers by E Jacotot
This network shows the impact of papers produced by E Jacotot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E Jacotot. The network helps show where E Jacotot may publish in the future.
Co-authors
The 25 scholars most cited alongside E Jacotot, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2000 | 196 | |
| 2 | Apoptosis induction by the photosensitizer verteporfin: identification of mitochondrial adenine nucleotide translocator as a critical target. | 2001 | 69 |
| 3 | 1999 | 67 | |
| 4 | 2012 | 60 | |
| 5 | Apoptosis and cell cycle: distinct checkpoints with overlapping upstream control. | 2000 | 23 |
About E Jacotot
E Jacotot is a scholar working on Molecular Biology, Infectious Diseases, Virology, Pulmonary and Respiratory Medicine and Clinical Biochemistry, having authored 5 papers that have together received 415 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (2 papers), Metabolism and Genetic Disorders (1 paper), Cell death mechanisms and regulation (1 paper), HIV Research and Treatment (1 paper), Alzheimer's disease research and treatments (1 paper), HIV/AIDS drug development and treatment (1 paper), Retinoids in leukemia and cellular processes (1 paper) and ATP Synthase and ATPases Research (1 paper). The work is most often cited by research in Virology (46 citations), Clinical Biochemistry (32 citations), Molecular Biology (290 citations), Aging (6 citations) and Cancer Research (37 citations). E Jacotot has collaborated with scholars based in France and United Kingdom. Frequent co-authors include Guido Kroemer, Catherine Brenner, Helena L.A. Vieira, D. Haouzi, Chahrazed El Hamel, Nathanaël Larochette, Zhihua Xie, L Ravagnan, Paola Costantini and Santos A. Susín. Their work appears in journals such as APOPTOSIS, Cell Death and Disease, Cell Death and Differentiation and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.