E. E. Calle

1.9k citations
10 papers · 1.3k · h-index 10

Impact in

Papers in

    • Inflammatory mediators and NSAID effects 3
    • Cancer Genomics and Diagnostics 1
    • Cancer, Lipids, and Metabolism 1

E. E. Calle

10 papers receiving 1.2k citations

Peers

E. E. Calle
Comparison fields: 5 of 104
  • Pharmacology 224
  • Cancer Research 186
  • Oncology 335
  • Reproductive Medicine 96
  • Neurology 90
Replace David J. Hunter with:
David J. Hunter United States
Andrea Iorga United States
Kazuko Arakawa United States
Weiqiong Gu China
J.R.L. Masarei Australia
Benedetta Bendinelli Italy
Ole‐Martin Fuskevåg Norway
Tevfik Sabuncu Türkiye
Nikolaos Spyrou United States
L N Kolonel United States
E. E. Calle relative to David J. Hunter United States David J. Hunter's profile →
Citations per field
00.5×2×3×4×4.7×
David J. Hunter · 1×
Citations per year

Countries citing papers authored by E. E. Calle

Since Specialization
Citations

This map shows the geographic impact of E. E. Calle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. E. Calle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. E. Calle more than expected).

Fields of papers citing papers by E. E. Calle

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. E. Calle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. E. Calle. The network helps show where E. E. Calle may publish in the future.

Co-authors

The 25 scholars most cited alongside E. E. Calle, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E. E. Calle Line = papers co-authored together E. E. Calle links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 1992334
2 2007251
3 2012187
4 2007149
5 2002109
6 200484
7 200465
8 200941
9 199941
10 200728

About E. E. Calle

E. E. Calle is a scholar working on Pharmacology, Cancer Research, Molecular Biology, Health and Clinical Psychology, having authored 10 papers that have together received 1.3k indexed citations. Recurring topics across this work include Inflammatory mediators and NSAID effects (3 papers), Peroxisome Proliferator-Activated Receptors (1 paper), Epigenetics and DNA Methylation (1 paper), Cancer Genomics and Diagnostics (1 paper), Suicide and Self-Harm Studies (1 paper), Gun Ownership and Violence Research (1 paper), BRCA gene mutations in cancer (1 paper) and Cancer, Lipids, and Metabolism (1 paper). The work is most often cited by research in Pharmacology (224 citations), Cancer Research (186 citations), Oncology (335 citations), Reproductive Medicine (96 citations) and Neurology (90 citations). E. E. Calle has collaborated with scholars based in United States, France and Canada. Frequent co-authors include Michael J. Thun, S. Jane Henley, Eric J. Jacobs, T. Byers, Carmen Rodríguez, Elizabeth B. Bain, W. Dana Flanders, C. W. Heath, Ralph J. Coates and Paolo Boffetta. Their work appears in journals such as JNCI Journal of the National Cancer Institute, American Journal of Epidemiology, The Lancet Oncology, CA A Cancer Journal for Clinicians and Carcinogenesis.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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