Debora Steiner
Impact in
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- Amyotrophic Lateral Sclerosis Research
- Neuroinflammation and Neurodegeneration Mechanisms
Papers in
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- Ion channel regulation and function 3
- Receptor Mechanisms and Signaling 3
- Pluripotent Stem Cells Research 2
- CRISPR and Genetic Engineering 2
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- Neuropeptides and Animal Physiology 4
- Co-authors
- Ester Schallmach (5 shared papers)Zvi Vogel (5 shared papers)Benjamin Reubinoff (4 shared papers)Yaniv Gil (4 shared papers)Tikva Turetsky (3 shared papers)Eva L. Feldman (1 shared paper)Oded Behar (1 shared paper)Israel Ben‐Dor (1 shared paper)
- Journals
- Stem Cell Reports (2 papers)Journal of Molecular Neuroscience (2 papers)Cellular Signalling (1 paper)Journal of Virology (1 paper)Neuropharmacology (1 paper)
- Partner nations
- IsraelUnited States
In The Last Decade
Debora Steiner
9 papers receiving 246 citations
Peers
Comparison fields: 5 of 49
- Neurology 47
- Neurology 81
- Developmental Neuroscience 18
- Cellular and Molecular Neuroscience 78
- Genetics 41
Countries citing papers authored by Debora Steiner
This map shows the geographic impact of Debora Steiner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Debora Steiner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Debora Steiner more than expected).
Fields of papers citing papers by Debora Steiner
This network shows the impact of papers produced by Debora Steiner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Debora Steiner. The network helps show where Debora Steiner may publish in the future.
Co-authors
The 25 scholars most cited alongside Debora Steiner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 127 | |
| 2 | 2005 | 36 | |
| 3 | 2022 | 24 | |
| 4 | 2006 | 16 | |
| 5 | 2015 | 15 | |
| 6 | 2005 | 14 | |
| 7 | 2005 | 8 | |
| 8 | 2022 | 6 | |
| 9 | 2006 | 5 |
About Debora Steiner
Debora Steiner is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Neurology, Physiology and Genetics, having authored 9 papers that have together received 251 indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (4 papers), Ion channel regulation and function (3 papers), Receptor Mechanisms and Signaling (3 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Pluripotent Stem Cells Research (2 papers), CRISPR and Genetic Engineering (2 papers), Cytomegalovirus and herpesvirus research (1 paper) and Pain Mechanisms and Treatments (1 paper). The work is most often cited by research in Neurology (47 citations), Neurology (81 citations), Developmental Neuroscience (18 citations), Cellular and Molecular Neuroscience (78 citations) and Genetics (41 citations). Debora Steiner has collaborated with scholars based in Israel and United States. Frequent co-authors include Ester Schallmach, Zvi Vogel, Benjamin Reubinoff, Yaniv Gil, Tikva Turetsky, Eva L. Feldman, Oded Behar, Israel Ben‐Dor, Sahar Sweetat and Vitali Belzer. Their work appears in journals such as Stem Cell Reports, Journal of Molecular Neuroscience, Cellular Signalling, Journal of Virology and Neuropharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.