David M. Wiley
Impact in
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- Zebrafish Biomedical Research Applications
- Hippo pathway signaling and YAP/TAZ
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- Angiogenesis and VEGF in Cancer
- Congenital heart defects research
- TGF-β signaling in diseases
- Kruppel-like factors research
Papers in
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- Angiogenesis and VEGF in Cancer 4
- Kruppel-like factors research 2
- Congenital heart defects research 2
- Connective Tissue Growth Factor Research 1
- TGF-β signaling in diseases 1
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- Zebrafish Biomedical Research Applications 2
- Co-authors
- Victoria L. Bautch (4 shared papers)John C. Chappell (2 shared papers)Jijun Hao (1 shared paper)Jun‐Dae Kim (1 shared paper)Charles C. Hong (1 shared paper)Suk‐Won Jin (1 shared paper)Zhongming Chen (1 shared paper)Daniel T. Sweet (1 shared paper)
- Journals
- Seminars in Cell and Developmental Biology (2 papers)Cells Tissues Organs (1 paper)Nature Cell Biology (1 paper)Blood (1 paper)
- Partner nations
- United StatesAustraliaRussia
In The Last Decade
David M. Wiley
5 papers receiving 312 citations
Peers
Comparison fields: 5 of 63
- Cell Biology 100
- Molecular Biology 215
- Cancer Research 36
- Immunology and Allergy 14
- Modeling and Simulation 7
Countries citing papers authored by David M. Wiley
This map shows the geographic impact of David M. Wiley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Wiley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Wiley more than expected).
Fields of papers citing papers by David M. Wiley
This network shows the impact of papers produced by David M. Wiley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Wiley. The network helps show where David M. Wiley may publish in the future.
Co-authors
The 9 scholars most cited alongside David M. Wiley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 161 | |
| 2 | 2011 | 66 | |
| 3 | 2011 | 40 | |
| 4 | 2011 | 30 | |
| 5 | 2011 | 22 |
About David M. Wiley
David M. Wiley is a scholar working on Molecular Biology, Cell Biology, Cellular and Molecular Neuroscience, Immunology and Allergy and Infectious Diseases, having authored 5 papers that have together received 319 indexed citations. Recurring topics across this work include Angiogenesis and VEGF in Cancer (4 papers), Zebrafish Biomedical Research Applications (2 papers), Kruppel-like factors research (2 papers), Congenital heart defects research (2 papers), Connective Tissue Growth Factor Research (1 paper), Cell Adhesion Molecules Research (1 paper), Axon Guidance and Neuronal Signaling (1 paper) and TGF-β signaling in diseases (1 paper). The work is most often cited by research in Cell Biology (100 citations), Molecular Biology (215 citations), Cancer Research (36 citations), Immunology and Allergy (14 citations) and Modeling and Simulation (7 citations). David M. Wiley has collaborated with scholars based in United States, Australia and Russia. Frequent co-authors include Victoria L. Bautch, John C. Chappell, Jijun Hao, Jun‐Dae Kim, Charles C. Hong, Suk‐Won Jin, Zhongming Chen, Daniel T. Sweet and Ellie Tzima. Their work appears in journals such as Seminars in Cell and Developmental Biology, Cells Tissues Organs, Nature Cell Biology and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.