David Broadbent
Impact in
-
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
Papers in
-
- Receptor Mechanisms and Signaling 2
- Epigenetics and DNA Methylation 2
- DNA Repair Mechanisms 1
- Ubiquitin and proteasome pathways 1
- Enzyme Catalysis and Immobilization 1
-
- Autophagy in Disease and Therapy 4
- Co-authors
- Jens C. Schmidt (5 shared papers)A. G. MORTON (1 shared paper)Carlo Barnaba (3 shared papers)Gloria I. Perez (3 shared papers)I.C. Tommerup (1 shared paper)Vajira Weeresekara (1 shared paper)David M. Thomson (1 shared paper)John T. Prince (1 shared paper)
- Journals
- The Journal of Cell Biology (2 papers)Advances in immunology (2 papers)Annals of Botany (1 paper)Molecular Biology of the Cell (1 paper)Nature (1 paper)
- Partner nations
- United StatesUnited KingdomIndia
In The Last Decade
David Broadbent
13 papers receiving 279 citations
Peers
Comparison fields: 5 of 69
- Cell Biology 59
- Physiology 16
- Epidemiology 103
- Pharmacology 40
- Molecular Biology 159
Countries citing papers authored by David Broadbent
This map shows the geographic impact of David Broadbent's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Broadbent with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Broadbent more than expected).
Fields of papers citing papers by David Broadbent
This network shows the impact of papers produced by David Broadbent. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Broadbent. The network helps show where David Broadbent may publish in the future.
Co-authors
The 24 scholars most cited alongside David Broadbent, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2014 | 95 | |
| 2 | 2023 | 51 | |
| 3 | 1955 | 41 | |
| 4 | 1975 | 26 | |
| 5 | 2017 | 23 | |
| 6 | 2024 | 16 | |
| 7 | 2023 | 14 | |
| 8 | 2022 | 13 | |
| 9 | 2023 | 6 | |
| 10 | 2017 | 4 | |
| 11 | 1966 | 3 | |
| 12 | 1958 | 3 | |
| 13 | 2024 | 2 |
About David Broadbent
David Broadbent is a scholar working on Molecular Biology, Epidemiology, Surgery, Pharmacology and Oncology, having authored 13 papers that have together received 297 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (4 papers), Pancreatic function and diabetes (3 papers), Receptor Mechanisms and Signaling (2 papers), Epigenetics and DNA Methylation (2 papers), DNA Repair Mechanisms (1 paper), Ubiquitin and proteasome pathways (1 paper), Plant Physiology and Cultivation Studies (1 paper) and Enzyme Catalysis and Immobilization (1 paper). The work is most often cited by research in Cell Biology (59 citations), Physiology (16 citations), Epidemiology (103 citations), Pharmacology (40 citations) and Molecular Biology (159 citations). David Broadbent has collaborated with scholars based in United States, United Kingdom and India. Frequent co-authors include Jens C. Schmidt, A. G. MORTON, Carlo Barnaba, Gloria I. Perez, I.C. Tommerup, Vajira Weeresekara, David M. Thomson, John T. Prince, Andrew D. Mathis and Joshua L. Andersen. Their work appears in journals such as The Journal of Cell Biology, Advances in immunology, Annals of Botany, Molecular Biology of the Cell and Nature.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.