David A. Ford
Impact in
- Biochemistry top 0.2%
- Lipid metabolism and biosynthesis
- Immunology top 2%
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms
Papers in
-
- Metabolomics and Mass Spectrometry Studies 18
- Protein Kinase Regulation and GTPase Signaling 14
- Peroxisome Proliferator-Activated Receptors 13
- Immunology 43
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 31
- Co-authors
- Carolyn J. Albert (55 shared papers)Richard W. Gross (15 shared papers)Fong‐Fu Hsu (12 shared papers)Stanley L. Hazen (7 shared papers)Jeffrey E. Saffitz (2 shared papers)Arun K. Thukkani (8 shared papers)Bo Wang (8 shared papers)Peter Tontonoz (7 shared papers)
- Journals
- Journal of Lipid Research (16 papers)Journal of Biological Chemistry (16 papers)American Journal of Physiology-Heart and Circulatory Physiology (7 papers)FEBS Letters (5 papers)The FASEB Journal (5 papers)
- Partner nations
- United StatesSpainCanada
In The Last Decade
David A. Ford
167 papers receiving 6.6k citations
David A. Ford's Hit Papers
Peers
Comparison fields: 5 of 140
- Biochemistry 820
- Immunology 1.4k
- Cancer Research 848
- Physiology 1.2k
- Molecular Biology 3.3k
Countries citing papers authored by David A. Ford
This map shows the geographic impact of David A. Ford's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Ford with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Ford more than expected).
Fields of papers citing papers by David A. Ford
This network shows the impact of papers produced by David A. Ford. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Ford. The network helps show where David A. Ford may publish in the future.
Co-authors
The 25 scholars most cited alongside David A. Ford, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 170 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | A novel mouse model of lipotoxic cardiomyopathy Hit paper breakdown → | 2001 | 637 |
| 2 | 2019 | 298 | |
| 3 | 2013 | 252 | |
| 4 | 2018 | 236 | |
| 5 | 2021 | 210 | |
| 6 | 2018 | 195 | |
| 7 | 2015 | 173 | |
| 8 | 2003 | 165 | |
| 9 | 1991 | 157 | |
| 10 | 2012 | 139 | |
| 11 | 2017 | 108 | |
| 12 | 1991 | 104 | |
| 13 | 1989 | 100 | |
| 14 | 2016 | 97 | |
| 15 | 2001 | 90 | |
| 16 | 2002 | 80 | |
| 17 | 2003 | 80 | |
| 18 | 2016 | 76 | |
| 19 | 1989 | 76 | |
| 20 | 2003 | 73 |
About David A. Ford
David A. Ford is a scholar working on Molecular Biology, Immunology, Physiology, Biochemistry and Epidemiology, having authored 170 papers that have together received 6.7k indexed citations. Recurring topics across this work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (31 papers), Nitric Oxide and Endothelin Effects (23 papers), Metabolomics and Mass Spectrometry Studies (18 papers), Lipid metabolism and biosynthesis (17 papers), Protein Kinase Regulation and GTPase Signaling (14 papers), Peroxisome Proliferator-Activated Receptors (13 papers), Eicosanoids and Hypertension Pharmacology (13 papers) and Adipose Tissue and Metabolism (12 papers). The work is most often cited by research in Biochemistry (820 citations), Immunology (1.4k citations), Cancer Research (848 citations), Physiology (1.2k citations) and Molecular Biology (3.3k citations). David A. Ford has collaborated with scholars based in United States, Spain and Canada. Frequent co-authors include Carolyn J. Albert, Richard W. Gross, Fong‐Fu Hsu, Stanley L. Hazen, Jeffrey E. Saffitz, Arun K. Thukkani, Bo Wang, Peter Tontonoz, Xin Rong and Jean E. Schaffer. Their work appears in journals such as Journal of Lipid Research, Journal of Biological Chemistry, American Journal of Physiology-Heart and Circulatory Physiology, FEBS Letters and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.