Daniel Rueda

56 papers receiving 2.5k citations

Peers

Daniel Rueda
Comparison fields: 5 of 76
  • Pharmacology 1.2k
  • Pathology and Forensic Medicine 484
  • Cancer Research 282
  • Developmental Neuroscience 42
  • Oncology 208
Replace Glenn S. Belinsky with:
Glenn S. Belinsky United States
Mai P. Nghiem Canada
Iris Lavon Israel
Bernard Rothhut France
Alberto Pérez‐Rosado Spain
Riccardo Taulli Italy
David K. Bol United States
Carmen Page United States
Adele M. Pauley United States
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Citations per year

Countries citing papers authored by Daniel Rueda

Since Specialization
Citations

This map shows the geographic impact of Daniel Rueda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Rueda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Rueda more than expected).

Fields of papers citing papers by Daniel Rueda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Rueda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Rueda. The network helps show where Daniel Rueda may publish in the future.

Co-authors

The 25 scholars most cited alongside Daniel Rueda, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Daniel Rueda Line = papers co-authored together Daniel Rueda links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 56 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Inhibition of glioma growth in vivo by selective activation of the CB(2) cannabinoid receptor.
2001382
2 2008261
3 2002216
4 2000173
5 2002166
6 1998164
7 1999147
8 2021106
9 201590
10 200188
11 200677
12 201373
13 200742
14 201942
15 200535
16 201731
17 201528
18 201928
19 201928
20 202225

About Daniel Rueda

Daniel Rueda is a scholar working on Pathology and Forensic Medicine, Pharmacology, Cancer Research, Oncology and Genetics, having authored 56 papers that have together received 2.6k indexed citations. Recurring topics across this work include Genetic factors in colorectal cancer (30 papers), Cannabis and Cannabinoid Research (8 papers), NF-κB Signaling Pathways (3 papers), Colorectal Cancer Treatments and Studies (3 papers), Myeloproliferative Neoplasms: Diagnosis and Treatment (2 papers), Immunodeficiency and Autoimmune Disorders (2 papers), Colorectal Cancer Screening and Detection (2 papers) and BRCA gene mutations in cancer (2 papers). The work is most often cited by research in Pharmacology (1.2k citations), Pathology and Forensic Medicine (484 citations), Cancer Research (282 citations), Developmental Neuroscience (42 citations) and Oncology (208 citations). Daniel Rueda has collaborated with scholars based in Spain, United States and Germany. Frequent co-authors include Ismael Galve‐Roperh, Manuel Guzmán, Cristina Sánchez, Guillermo Velasco, Teresa Gómez del Pulgar, Alberto Martı́nez-Serrano, Amador Haro, Margot Thome, César Corbacho and Maria L. DE CEBALLOS. Their work appears in journals such as Molecular Pharmacology, Gastroenterology, Clinical Genetics, British Journal of Cancer and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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