Daniel K. Stringer
Impact in
- Cell Biology top 10%
- Cellular transport and secretion
- Endoplasmic Reticulum Stress and Disease
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- Ubiquitin and proteasome pathways
- Protein Degradation and Inhibitors
- Glycosylation and Glycoproteins Research
Papers in
-
- Ubiquitin and proteasome pathways 4
- Glycosylation and Glycoproteins Research 1
- Oncology 5
- Cancer-related Molecular Pathways 5
- Co-authors
- Robert C. Piper (5 shared papers)Judith Souphron (1 shared paper)Daniel C. Scott (1 shared paper)Hari Kamadurai (1 shared paper)Brenda A. Schulman (1 shared paper)David M. Duda (1 shared paper)Darcie J. Miller (1 shared paper)Chris MacDonald (2 shared papers)
- Journals
- Journal of Biological Chemistry (2 papers)The Journal of Cell Biology (1 paper)Oncotarget (1 paper)EMBO Reports (1 paper)Traffic (1 paper)
- Partner nations
- United StatesFranceItaly
In The Last Decade
Daniel K. Stringer
9 papers receiving 593 citations
Peers
Comparison fields: 5 of 62
- Cell Biology 181
- Molecular Biology 495
- Oncology 152
- Physiology 21
- Epidemiology 123
Countries citing papers authored by Daniel K. Stringer
This map shows the geographic impact of Daniel K. Stringer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel K. Stringer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel K. Stringer more than expected).
Fields of papers citing papers by Daniel K. Stringer
This network shows the impact of papers produced by Daniel K. Stringer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel K. Stringer. The network helps show where Daniel K. Stringer may publish in the future.
Co-authors
The 25 scholars most cited alongside Daniel K. Stringer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 240 | |
| 2 | 2011 | 116 | |
| 3 | 2012 | 66 | |
| 4 | 2003 | 64 | |
| 5 | 2011 | 38 | |
| 6 | 2015 | 27 | |
| 7 | 2016 | 25 | |
| 8 | 2011 | 21 | |
| 9 | 2002 | 2 |
About Daniel K. Stringer
Daniel K. Stringer is a scholar working on Molecular Biology, Oncology, Epidemiology, Cell Biology and Physiology, having authored 9 papers that have together received 599 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (5 papers), Ubiquitin and proteasome pathways (4 papers), Autophagy in Disease and Therapy (3 papers), Cellular transport and secretion (3 papers), interferon and immune responses (1 paper), Glycosylation and Glycoproteins Research (1 paper), NF-κB Signaling Pathways (1 paper) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in Cell Biology (181 citations), Molecular Biology (495 citations), Oncology (152 citations), Physiology (21 citations) and Epidemiology (123 citations). Daniel K. Stringer has collaborated with scholars based in United States, France and Italy. Frequent co-authors include Robert C. Piper, Judith Souphron, Daniel C. Scott, Hari Kamadurai, Brenda A. Schulman, David M. Duda, Darcie J. Miller, Chris MacDonald, Rolf Jakobi and Nicholas J. Buchkovich. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology, Oncotarget, EMBO Reports and Traffic.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.