Daha Mr
Impact in
- Nephrology top 2%
- Renal Diseases and Glomerulopathies
- Immunology top 5%
- Complement system in diseases
Papers in
- Immunology 36
- Complement system in diseases 21
- Immunodeficiency and Autoimmune Disorders 8
- Hematology 13
- Blood groups and transfusion 9
- Co-authors
- van Es La (29 shared papers)Fearon Dt (4 shared papers)E. Christiaan Hagen (2 shared papers)Austen Kf (3 shared papers)Kat Austen (1 shared paper)S. Lobatto (2 shared papers)Breedveld Fc (3 shared papers)LA Ginsel (3 shared papers)
- Journals
- Blood (5 papers)Transplant Immunology (1 paper)Clinical & Experimental Immunology (1 paper)Lupus (1 paper)European Journal of Gastroenterology & Hepatology (1 paper)
- Partner nations
- NetherlandsDenmarkUnited States
In The Last Decade
Daha Mr
58 papers receiving 906 citations
Peers
Comparison fields: 5 of 71
- Nephrology 280
- Immunology 513
- Hematology 195
- Transplantation 35
- Genetics 132
Countries citing papers authored by Daha Mr
This map shows the geographic impact of Daha Mr's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daha Mr with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daha Mr more than expected).
Fields of papers citing papers by Daha Mr
This network shows the impact of papers produced by Daha Mr. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daha Mr. The network helps show where Daha Mr may publish in the future.
Co-authors
The 25 scholars most cited alongside Daha Mr, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 59 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 1993 | 109 | |
| 2 | 1993 | 96 | |
| 3 | C3 nephritic factor (C3NeF): stabilization of fluid phase and cell-bound alternative pathway convertase. | 1976 | 83 |
| 4 | Antibody, immune complexes, and complement activity fluctuations in kittens with experimentally induced feline infectious peritonitis. | 1982 | 59 |
| 5 | Heterogeneity, polypeptide chain composition and antigenic reactivity of C3 nephritic factor. | 1978 | 51 |
| 6 | Clearance of soluble aggregates of human immunoglobulin G in healthy volunteers and chimpanzees. | 1987 | 39 |
| 7 | The incorporation of C3 nephritic factor (C3NeF) into a stabilized C3 convertase, C3bBb(C3NeF), and its release after decay of convertase function. | 1977 | 37 |
| 8 | The role of the c-ANCA antigen in the pathogenesis of Wegener's granulomatosis. A hypothesis based on both humoral and cellular mechanisms. | 1990 | 35 |
| 9 | Characterization of anti-endothelial antibodies in patients with rheumatoid arthritis complicated by vasculitis. | 1992 | 33 |
| 10 | Histomorphometric correlates of renal failure in IgA nephropathy. | 1998 | 33 |
| 11 | Further evidence for the antibody nature of C3 nephritic factor (C3NeF). | 1979 | 28 |
| 12 | IgA antibodies directed against cytoplasmic antigens of polymorphonuclear leukocytes in patients with Henoch-Schoenlein purpura. | 1987 | 28 |
| 13 | Regulation of the C3 nephritic factor stabilized C3/C5 convertase of complement by purified human erythrocyte C3b receptor. | 1982 | 26 |
| 14 | Enhanced alternative complement pathway-dependent degradation of soluble immunoglobulin aggregates by macrophages. | 1981 | 25 |
| 15 | Immunoelectron microscopic visualization of the transcytosis of low density lipoproteins in perfused rat arteries. | 1989 | 24 |
| 16 | 1991 | 22 | |
| 17 | Epithelial- and endothelial-cell specificity of renal graft infiltrating T cells. | 1998 | 20 |
| 18 | The role of cellular Fc and C3 receptors on the complement-dependent degradation of stable soluble immunoglobulin aggregates by normal and trypsin-treated peritoneal macrophages. | 1982 | 19 |
| 19 | Enhanced degradation of soluble immune complexes by guinea-pig peritoneal macrophages in the presence of complement. | 1981 | 13 |
| 20 | IgM, IgG, and IgA antibodies in human sera directed against porcine islets of Langerhans. | 1993 | 13 |
About Daha Mr
Daha Mr is a scholar working on Immunology, Hematology, Molecular Biology, Radiology, Nuclear Medicine and Imaging and Rheumatology, having authored 59 papers that have together received 995 indexed citations. Recurring topics across this work include Complement system in diseases (21 papers), Monoclonal and Polyclonal Antibodies Research (11 papers), Renal Diseases and Glomerulopathies (10 papers), Blood groups and transfusion (9 papers), Immunodeficiency and Autoimmune Disorders (8 papers), Erythrocyte Function and Pathophysiology (6 papers), Vasculitis and related conditions (6 papers) and Systemic Lupus Erythematosus Research (6 papers). The work is most often cited by research in Nephrology (280 citations), Immunology (513 citations), Hematology (195 citations), Transplantation (35 citations) and Genetics (132 citations). Daha Mr has collaborated with scholars based in Netherlands, Denmark and United States. Frequent co-authors include van Es La, Fearon Dt, E. Christiaan Hagen, Austen Kf, Kat Austen, S. Lobatto, Breedveld Fc, LA Ginsel, Liesbeth E. Jonges and Jack Fransen. Their work appears in journals such as Blood, Transplant Immunology, Clinical & Experimental Immunology, Lupus and European Journal of Gastroenterology & Hepatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.