Chenchao Ma
Impact in
- Cancer Research top 5%
- Cancer-related molecular mechanisms research
- MicroRNA in disease regulation
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- RNA modifications and cancer
- Circular RNAs in diseases
- Extracellular vesicles in disease
- RNA Research and Splicing
- Ubiquitin and proteasome pathways
Papers in
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- Protein Tyrosine Phosphatases 1
- Hedgehog Signaling Pathway Studies 1
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- MicroRNA in disease regulation 2
- Co-authors
- Kaixing Ai (5 shared papers)Kate Nong (4 shared papers)Zhou Yuan (3 shared papers)Hongda Zhu (3 shared papers)Xinyu Huang (3 shared papers)Weiwei Wang (2 shared papers)Bo Wu (3 shared papers)Yueqing Bai (3 shared papers)
- Journals
- Tumor Biology (2 papers)American Journal of Physiology-Gastrointestinal and Liver Physiology (1 paper)Cytotherapy (1 paper)Journal of Biological Chemistry (1 paper)Journal of Experimental & Clinical Cancer Research (1 paper)
- Partner nations
- ChinaUnited StatesSouth Korea
In The Last Decade
Chenchao Ma
7 papers receiving 533 citations
Peers
Comparison fields: 5 of 61
- Cancer Research 386
- Molecular Biology 469
- Genetics 29
- Oncology 65
- Hepatology 18
Countries citing papers authored by Chenchao Ma
This map shows the geographic impact of Chenchao Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chenchao Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chenchao Ma more than expected).
Fields of papers citing papers by Chenchao Ma
This network shows the impact of papers produced by Chenchao Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chenchao Ma. The network helps show where Chenchao Ma may publish in the future.
Co-authors
The 25 scholars most cited alongside Chenchao Ma, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2014 | 236 | |
| 2 | 2016 | 137 | |
| 3 | 2014 | 77 | |
| 4 | 2014 | 41 | |
| 5 | 2021 | 27 | |
| 6 | 2020 | 12 | |
| 7 | 2020 | 7 |
About Chenchao Ma
Chenchao Ma is a scholar working on Molecular Biology, Cancer Research, Surgery, Genetics and Pathology and Forensic Medicine, having authored 7 papers that have together received 537 indexed citations. Recurring topics across this work include MicroRNA in disease regulation (2 papers), Mesenchymal stem cell research (1 paper), Diet, Metabolism, and Disease (1 paper), Protein Tyrosine Phosphatases (1 paper), Galectins and Cancer Biology (1 paper), Cancer-related Molecular Pathways (1 paper), Pancreatitis Pathology and Treatment (1 paper) and Hedgehog Signaling Pathway Studies (1 paper). The work is most often cited by research in Cancer Research (386 citations), Molecular Biology (469 citations), Genetics (29 citations), Oncology (65 citations) and Hepatology (18 citations). Chenchao Ma has collaborated with scholars based in China, United States and South Korea. Frequent co-authors include Kaixing Ai, Kate Nong, Zhou Yuan, Hongda Zhu, Xinyu Huang, Weiwei Wang, Bo Wu, Yueqing Bai, Bin Hu and Xin Niu. Their work appears in journals such as Tumor Biology, American Journal of Physiology-Gastrointestinal and Liver Physiology, Cytotherapy, Journal of Biological Chemistry and Journal of Experimental & Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.