Charles E. Mays

1.4k citations
34 papers · 1.1k · h-index 18

Impact in

Papers in

    • Prion Diseases and Protein Misfolding 25
    • RNA regulation and disease 3
    • RNA Research and Splicing 2
    • Neurological diseases and metabolism 15

Charles E. Mays

32 papers receiving 1.0k citations

Peers

Charles E. Mays
Comparison fields: 5 of 99
  • Neurology 273
  • Developmental Neuroscience 86
  • Nutrition and Dietetics 174
  • Molecular Biology 725
  • Physiology 221
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Mauricio Torres United States
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Huan Zhang China
Nicolle Queiroz‐Hazarbassanov Brazil
Bradley R. Groveman United States
Naoto Matsuda Japan
Rosalina Gavı́n Spain
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Citations per field
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Citations per year

Countries citing papers authored by Charles E. Mays

Since Specialization
Citations

This map shows the geographic impact of Charles E. Mays's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles E. Mays with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles E. Mays more than expected).

Fields of papers citing papers by Charles E. Mays

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles E. Mays. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles E. Mays. The network helps show where Charles E. Mays may publish in the future.

Co-authors

The 25 scholars most cited alongside Charles E. Mays, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Charles E. Mays Line = papers co-authored together Charles E. Mays links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 34 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2018274
2 2018133
3 1975103
4 201459
5 197355
6 200943
7 201534
8 201133
9 201527
10 201926
11 201125
12 201025
13 201623
14 201323
15 200922
16 197322
17 201119
18 201318
19 201817
20 200817

About Charles E. Mays

Charles E. Mays is a scholar working on Molecular Biology, Neurology, Nutrition and Dietetics, Virology and Ecology, having authored 34 papers that have together received 1.1k indexed citations. Recurring topics across this work include Prion Diseases and Protein Misfolding (25 papers), Neurological diseases and metabolism (15 papers), Trace Elements in Health (8 papers), HIV Research and Treatment (4 papers), RNA regulation and disease (3 papers), Amphibian and Reptile Biology (3 papers), Wildlife Ecology and Conservation (2 papers) and RNA Research and Splicing (2 papers). The work is most often cited by research in Neurology (273 citations), Developmental Neuroscience (86 citations), Nutrition and Dietetics (174 citations), Molecular Biology (725 citations) and Physiology (221 citations). Charles E. Mays has collaborated with scholars based in United States, Canada and South Korea. Frequent co-authors include Claudio Soto, Max A. Nickerson, Chongsuk Ryou, Enrique Armijo, Andrea Becerra-Calixto, César González, Javiera Bravo‐Alegria, Harold A. Dundee, David Westaway and Inés Moreno‐González. Their work appears in journals such as Biomaterials, PLoS Pathogens, Copeia, Brain Research and The FASEB Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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