Celine Be

719 citations
12 papers · 527 · h-index 10

Impact in

    • Protein Degradation and Inhibitors
    • Ubiquitin and proteasome pathways
    • Extracellular vesicles in disease
    • RNA modifications and cancer
    • Epigenetics and DNA Methylation

Papers in

Celine Be

10 papers receiving 519 citations

Peers

Celine Be
Comparison fields: 5 of 72
  • Molecular Biology 336
  • Structural Biology 7
  • Oncology 111
  • Hematology 37
  • Immunology 63
Replace Zhi Yong WU with:
Zhi Yong WU China
Sharon Shechter United States
Geo Stoyanov Derimanov United States
Michelle de la Vega United Kingdom
Stephanie Leimgruber United States
Magdalena B. Rother Netherlands
Cynthia B. J. Paulin Sweden
Ilaria Cerbara Italy
Kai‐Chen Wu Australia
Sohini Chakraborty India
Celine Be relative to Zhi Yong WU China Zhi Yong WU's profile →
Citations per field
00.5×2×2.6×
Zhi Yong WU · 1×
Citations per year

Countries citing papers authored by Celine Be

Since Specialization
Citations

This map shows the geographic impact of Celine Be's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Celine Be with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Celine Be more than expected).

Fields of papers citing papers by Celine Be

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Celine Be. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Celine Be. The network helps show where Celine Be may publish in the future.

Co-authors

The 25 scholars most cited alongside Celine Be, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Celine Be Line = papers co-authored together Celine Be links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 2019197
2 202086
3 201758
4 201944
5 201641
6 202339
7 201418
8 201916
9 201714
10 202213
11 20251
12 20250

About Celine Be

Celine Be is a scholar working on Immunology, Hematology, Molecular Biology, Oncology and Epidemiology, having authored 12 papers that have together received 527 indexed citations. Recurring topics across this work include Biochemical and Molecular Research (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), RNA Interference and Gene Delivery (2 papers), Trypanosoma species research and implications (2 papers), Renin-Angiotensin System Studies (1 paper), Immunodeficiency and Autoimmune Disorders (1 paper), Blood groups and transfusion (1 paper) and NF-κB Signaling Pathways (1 paper). The work is most often cited by research in Molecular Biology (336 citations), Structural Biology (7 citations), Oncology (111 citations), Hematology (37 citations) and Immunology (63 citations). Celine Be has collaborated with scholars based in Switzerland, United States and China. Frequent co-authors include Clemens Scheufler, Klemens Kaupmann, Klemens Hoegenauer, Samuel Hintermann, Luca Arista, Joerg A. Kallen, Christine Guntermann, David Orain, César Fernández Fernández and Ralph Tiedt. Their work appears in journals such as Nature Chemical Biology, Journal of Medicinal Chemistry, mAbs, ACS Medicinal Chemistry Letters and SLAS DISCOVERY.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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