Celia Vélez

54 papers receiving 698 citations

Peers

Celia Vélez
Comparison fields: 5 of 99
  • Biomaterials 128
  • Oncology 181
  • Molecular Medicine 26
  • Molecular Biology 321
  • Cancer Research 55
Replace Matin Chehelgerdi with:
Matin Chehelgerdi Iran
Takayuki Nakano Japan
Zhao Yang China
Chinmayee Sethy India
Soon‐Kyung Hwang South Korea
Xiaohua Luo China
Qianqian Wang China
Guan Jiang China
Celia Vélez relative to Matin Chehelgerdi Iran Matin Chehelgerdi's profile →
Citations per field
00.5×2.5×
Matin Chehelgerdi · 1×
Citations per year

Countries citing papers authored by Celia Vélez

Since Specialization
Citations

This map shows the geographic impact of Celia Vélez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Celia Vélez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Celia Vélez more than expected).

Fields of papers citing papers by Celia Vélez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Celia Vélez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Celia Vélez. The network helps show where Celia Vélez may publish in the future.

Co-authors

The 25 scholars most cited alongside Celia Vélez, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Celia Vélez Line = papers co-authored together Celia Vélez links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 55 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2012111
2 201292
3 201338
4 201533
5 197630
6 201628
7 199227
8 200324
9 200921
10 199519
11 199718
12 201915
13 201514
14 199814
15 201012
16 200712
17
Actinomycin D causes multidrug resistance and differentiation in a human rhabdomyosarcoma cell line.
199412
18 199911
19 201210
20 200210

About Celia Vélez

Celia Vélez is a scholar working on Molecular Biology, Oncology, Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine and Cell Biology, having authored 55 papers that have together received 717 indexed citations. Recurring topics across this work include Cardiomyopathy and Myosin Studies (9 papers), RNA Interference and Gene Delivery (8 papers), Cancer therapeutics and mechanisms (6 papers), Sarcoma Diagnosis and Treatment (6 papers), Cardiovascular Effects of Exercise (5 papers), Peptidase Inhibition and Analysis (5 papers), Nanoparticle-Based Drug Delivery (4 papers) and Immunotherapy and Immune Responses (4 papers). The work is most often cited by research in Biomaterials (128 citations), Oncology (181 citations), Molecular Medicine (26 citations), Molecular Biology (321 citations) and Cancer Research (55 citations). Celia Vélez has collaborated with scholars based in Spain, United States and Italy. Frequent co-authors include Consolación Melguizo, José Prados, Antonia Aránega, Pablo Álvarez, Juan Antonio Marchal, Raúl Ortíz, Houría Boulaiz, Esmeralda Carrillo, Fernando Rodríguez‐Serrano and José L. Arias. Their work appears in journals such as Cells Tissues Organs, American Journal of Veterinary Research, Journal of Cardiovascular Pharmacology, International Journal of Molecular Sciences and Life Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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