Caleb Pitcairn
Impact in
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- Circadian rhythm and melatonin
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- Parkinson's Disease Mechanisms and Treatments
Papers in
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- Parkinson's Disease Mechanisms and Treatments 6
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- Lysosomal Storage Disorders Research 4
- Co-authors
- Joseph R. Mazzulli (6 shared papers)Willayat Yousuf Wani (2 shared papers)Markus Heilig (4 shared papers)Estelle Barbier (4 shared papers)Leandro F. Vendruscolo (2 shared papers)Jenica D. Tapocik (2 shared papers)Jesse R. Schank (2 shared papers)Friederike Zunke (3 shared papers)
- Journals
- Journal of Neuroscience (2 papers)Translational Neurodegeneration (1 paper)Molecular Psychiatry (1 paper)Neuron (1 paper)Neuropsychopharmacology (1 paper)
- Partner nations
- United StatesSwedenGermany
In The Last Decade
Caleb Pitcairn
10 papers receiving 353 citations
Peers
Comparison fields: 5 of 63
- Endocrine and Autonomic Systems 44
- Neurology 88
- Cellular and Molecular Neuroscience 92
- Aging 7
- Cell Biology 63
Countries citing papers authored by Caleb Pitcairn
This map shows the geographic impact of Caleb Pitcairn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Caleb Pitcairn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Caleb Pitcairn more than expected).
Fields of papers citing papers by Caleb Pitcairn
This network shows the impact of papers produced by Caleb Pitcairn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Caleb Pitcairn. The network helps show where Caleb Pitcairn may publish in the future.
Co-authors
The 25 scholars most cited alongside Caleb Pitcairn, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 88 | |
| 2 | 2014 | 59 | |
| 3 | 2018 | 56 | |
| 4 | 2019 | 47 | |
| 5 | 2016 | 40 | |
| 6 | 2021 | 26 | |
| 7 | 2023 | 20 | |
| 8 | 2023 | 11 | |
| 9 | 2016 | 7 | |
| 10 | 2024 | 5 |
About Caleb Pitcairn
Caleb Pitcairn is a scholar working on Neurology, Physiology, Cellular and Molecular Neuroscience, Cell Biology and Molecular Biology, having authored 10 papers that have together received 359 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (6 papers), Lysosomal Storage Disorders Research (4 papers), Cellular transport and secretion (3 papers), Epigenetics and DNA Methylation (2 papers), Cannabis and Cannabinoid Research (2 papers), Autophagy in Disease and Therapy (2 papers), Nerve injury and regeneration (1 paper) and Prenatal Substance Exposure Effects (1 paper). The work is most often cited by research in Endocrine and Autonomic Systems (44 citations), Neurology (88 citations), Cellular and Molecular Neuroscience (92 citations), Aging (7 citations) and Cell Biology (63 citations). Caleb Pitcairn has collaborated with scholars based in United States, Sweden and Germany. Frequent co-authors include Joseph R. Mazzulli, Willayat Yousuf Wani, Markus Heilig, Estelle Barbier, Leandro F. Vendruscolo, Jenica D. Tapocik, Jesse R. Schank, Friederike Zunke, Kornel E. Schuebel and Qiaoping Yuan. Their work appears in journals such as Journal of Neuroscience, Translational Neurodegeneration, Molecular Psychiatry, Neuron and Neuropsychopharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.