C. Zang
Impact in
- Immunology and Allergy top 10%
- Cell Adhesion Molecules Research
-
- Cancer, Lipids, and Metabolism
- Cancer, Hypoxia, and Metabolism
Papers in
-
- Retinoids in leukemia and cellular processes 3
- Peroxisome Proliferator-Activated Receptors 2
- Glycosylation and Glycoproteins Research 1
- Cell death mechanisms and regulation 1
- Oncology 2
- Peptidase Inhibition and Analysis 1
- Co-authors
- K. Possinger (3 shared papers)Martin Fenner (2 shared papers)Elena Elstner (2 shared papers)David Heber (1 shared paper)H. Phillip Koeffler (1 shared paper)Elizabeth A. Williamson (1 shared paper)Justine Fritz (1 shared paper)Daniel L. Gustafson (1 shared paper)
- Journals
- Breast Cancer Research and Treatment (2 papers)Blood Cells Molecules and Diseases (1 paper)Annals of Oncology (1 paper)Leukemia (1 paper)Annals of Hematology (1 paper)
- Partner nations
- GermanyUnited StatesChina
In The Last Decade
C. Zang
7 papers receiving 389 citations
Peers
Comparison fields: 5 of 61
- Immunology and Allergy 59
- Cancer Research 109
- Molecular Biology 261
- Oncology 73
- Pharmacology 36
Countries citing papers authored by C. Zang
This map shows the geographic impact of C. Zang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. Zang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. Zang more than expected).
Fields of papers citing papers by C. Zang
This network shows the impact of papers produced by C. Zang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. Zang. The network helps show where C. Zang may publish in the future.
Co-authors
The 25 scholars most cited alongside C. Zang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2003 | 144 | |
| 2 | 2007 | 100 | |
| 3 | 2002 | 75 | |
| 4 | 1998 | 34 | |
| 5 | 2000 | 18 | |
| 6 | 2016 | 12 | |
| 7 | 2004 | 11 |
About C. Zang
C. Zang is a scholar working on Molecular Biology, Oncology, Cancer Research, Pharmacology and Epidemiology, having authored 7 papers that have together received 394 indexed citations. Recurring topics across this work include Retinoids in leukemia and cellular processes (3 papers), Peroxisome Proliferator-Activated Receptors (2 papers), Folate and B Vitamins Research (1 paper), Glycosylation and Glycoproteins Research (1 paper), Trypanosoma species research and implications (1 paper), Inflammatory mediators and NSAID effects (1 paper), Cell death mechanisms and regulation (1 paper) and Peptidase Inhibition and Analysis (1 paper). The work is most often cited by research in Immunology and Allergy (59 citations), Cancer Research (109 citations), Molecular Biology (261 citations), Oncology (73 citations) and Pharmacology (36 citations). C. Zang has collaborated with scholars based in Germany, United States and China. Frequent co-authors include K. Possinger, Martin Fenner, Elena Elstner, David Heber, H. Phillip Koeffler, Elizabeth A. Williamson, Justine Fritz, Daniel L. Gustafson, David J. McConkey and Sabine Eckhardt. Their work appears in journals such as Breast Cancer Research and Treatment, Blood Cells Molecules and Diseases, Annals of Oncology, Leukemia and Annals of Hematology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.