Boris Strilić
Impact in
- Oncology top 5%
- Cancer Cells and Metastasis
- Inflammatory Biomarkers in Disease Prognosis
- Lymphatic System and Diseases
- Cell Biology top 2%
- Hippo pathway signaling and YAP/TAZ
- Cellular Mechanics and Interactions
Papers in
-
- Hippo pathway signaling and YAP/TAZ 4
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- Angiogenesis and VEGF in Cancer 5
- Cell death mechanisms and regulation 4
- Receptor Mechanisms and Signaling 3
- Co-authors
- Stefan Offermanns (14 shared papers)Nina Wettschureck (8 shared papers)Eckhard Lammert (8 shared papers)Kishor Kumar Sivaraj (3 shared papers)Dagmar Schumacher (2 shared papers)Julián Albarrán-Juárez (3 shared papers)Lida Yang (2 shared papers)Manolis Pasparakis (1 shared paper)
- Journals
- The Journal of Experimental Medicine (3 papers)Cancer Cell (3 papers)Journal of Clinical Investigation (2 papers)PLoS ONE (2 papers)Molecular Cancer (1 paper)
- Partner nations
- GermanyUnited StatesCzechia
In The Last Decade
Boris Strilić
27 papers receiving 3.1k citations
Boris Strilić's Hit Papers
Peers
Comparison fields: 5 of 106
- Oncology 897
- Cell Biology 522
- Cancer Research 436
- Physiology 132
- Immunology and Allergy 159
Countries citing papers authored by Boris Strilić
This map shows the geographic impact of Boris Strilić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Strilić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Strilić more than expected).
Fields of papers citing papers by Boris Strilić
This network shows the impact of papers produced by Boris Strilić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Strilić. The network helps show where Boris Strilić may publish in the future.
Co-authors
The 25 scholars most cited alongside Boris Strilić, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 27 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Platelet-Derived Nucleotides Promote Tumor-Cell Transendothelial Migration and Metastasis via P2Y2 Receptor Hit paper breakdown → | 2013 | 529 |
| 2 | 2016 | 427 | |
| 3 | 2017 | 323 | |
| 4 | 2009 | 297 | |
| 5 | 2018 | 254 | |
| 6 | 2019 | 235 | |
| 7 | 2015 | 172 | |
| 8 | 2011 | 142 | |
| 9 | 2006 | 137 | |
| 10 | 2010 | 106 | |
| 11 | 2022 | 85 | |
| 12 | 2018 | 70 | |
| 13 | 2021 | 52 | |
| 14 | 2021 | 49 | |
| 15 | 2019 | 42 | |
| 16 | 2013 | 41 | |
| 17 | 2010 | 41 | |
| 18 | 2021 | 32 | |
| 19 | 2009 | 30 | |
| 20 | 2019 | 25 |
About Boris Strilić
Boris Strilić is a scholar working on Cell Biology, Molecular Biology, Immunology, Physiology and Oncology, having authored 27 papers that have together received 3.1k indexed citations. Recurring topics across this work include Angiogenesis and VEGF in Cancer (5 papers), Cell death mechanisms and regulation (4 papers), Hippo pathway signaling and YAP/TAZ (4 papers), Lymphatic System and Diseases (3 papers), Receptor Mechanisms and Signaling (3 papers), Erythrocyte Function and Pathophysiology (3 papers), Phagocytosis and Immune Regulation (3 papers) and Invertebrate Immune Response Mechanisms (2 papers). The work is most often cited by research in Oncology (897 citations), Cell Biology (522 citations), Cancer Research (436 citations), Physiology (132 citations) and Immunology and Allergy (159 citations). Boris Strilić has collaborated with scholars based in Germany, United States and Czechia. Frequent co-authors include Stefan Offermanns, Nina Wettschureck, Eckhard Lammert, Kishor Kumar Sivaraj, Dagmar Schumacher, Julián Albarrán-Juárez, Lida Yang, Manolis Pasparakis, Laurens Wachsmuth and Ulrike Müller. Their work appears in journals such as The Journal of Experimental Medicine, Cancer Cell, Journal of Clinical Investigation, PLoS ONE and Molecular Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.