Beth Muir

3.8k citations
11 papers · 2.4k · 2 hit papers · h-index 11

Impact in

Papers in

    • Wnt/β-catenin signaling in development and cancer 2
    • Developmental Biology and Gene Regulation 2
    • Cancer-related Molecular Pathways 2

Beth Muir

11 papers receiving 2.3k citations

Beth Muir's Hit Papers

Transforming properties of YAP , a candidate oncogene on the chromosome 11q22 amplicon 2006 · 750 citations
7500+7+14Years since publication250500750

Peers

Beth Muir
Comparison fields: 5 of 80
  • Cell Biology 701
  • Hepatology 222
  • Oncology 740
  • Cancer Research 337
  • Pulmonary and Respiratory Medicine 685
Replace Rajani Kanteti with:
Rajani Kanteti United States
Nila U. Parikh United States
Chenwei Li United States
Andrew B. Gladden United States
William Cruz‐Muñoz Canada
Jens Quant Austria
Silvia Benvenuti Italy
Jalila Adnane United States
Chun Jye Lim Singapore
Kwan Ho Tang Hong Kong
Beth Muir relative to Rajani Kanteti United States Rajani Kanteti's profile →
Citations per field
00.5×5.9×
Rajani Kanteti · 1×
Citations per year

Countries citing papers authored by Beth Muir

Since Specialization
Citations

This map shows the geographic impact of Beth Muir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth Muir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth Muir more than expected).

Fields of papers citing papers by Beth Muir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beth Muir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth Muir. The network helps show where Beth Muir may publish in the future.

Co-authors

The 25 scholars most cited alongside Beth Muir, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Beth Muir Line = papers co-authored together Beth Muir links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1
Transforming properties of YAP , a candidate oncogene on the chromosome 11q22 amplicon
Hit paper breakdown →
2006750
2
Epidermal Growth Factor Receptor Mutations and Gene Amplification in Non–Small-Cell Lung Cancer: Molecular Analysis of the IDEAL/INTACT Gefitinib Trials
Hit paper breakdown →
2005517
3 2006401
4 2006167
5 2006133
6 2005104
7 200798
8 200775
9 200760
10 200738
11 200632

About Beth Muir

Beth Muir is a scholar working on Molecular Biology, Oncology, Pathology and Forensic Medicine, Pulmonary and Respiratory Medicine and Cell Biology, having authored 11 papers that have together received 2.4k indexed citations. Recurring topics across this work include Liver physiology and pathology (2 papers), Cancer-related Molecular Pathways (2 papers), Lung Cancer Treatments and Mutations (2 papers), Wnt/β-catenin signaling in development and cancer (2 papers), Genetic factors in colorectal cancer (2 papers), Developmental Biology and Gene Regulation (2 papers), Hippo pathway signaling and YAP/TAZ (2 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (1 paper). The work is most often cited by research in Cell Biology (701 citations), Hepatology (222 citations), Oncology (740 citations), Cancer Research (337 citations) and Pulmonary and Respiratory Medicine (685 citations). Beth Muir has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Dennis C. Sgroi, Daniel A. Haber, Gromoslaw A. Smolen, Chu‐Xia Deng, Wenmei Li, Michael Overholtzer, Jianmin Zhang, Joan S. Brugge, Daphne W. Bell and Ross A. Okimoto. Their work appears in journals such as Proceedings of the National Academy of Sciences, Clinical Cancer Research, Journal of Clinical Oncology, The Journal of Immunology and Genes & Development.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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