Benjamin Schwalm

615 citations
6 papers · 46 · h-index 3

Impact in

    • Glioma Diagnosis and Treatment
    • Neuroblastoma Research and Treatments

Papers in

    • Glioma Diagnosis and Treatment 3
    • Hedgehog Signaling Pathway Studies 2
    • Genomics and Chromatin Dynamics 1
    • Chromatin Remodeling and Cancer 1

Benjamin Schwalm

6 papers receiving 44 citations

Peers

Benjamin Schwalm
Comparison fields: 5 of 21
  • Genetics 16
  • Neurology 8
  • Biotechnology 5
  • Biophysics 2
  • Molecular Biology 21
Replace Alicia Baumgartner with:
Alicia Baumgartner Austria
Jad Alshami Canada
Rodrigo T. Cartaxo United States
Janet Schmid United States
Esma Kerboua Algeria
Benjamin Carpentier France
Ivette Zelaya United States
Sina Kreth Germany
Stéphane Doucet Canada
Barbara Wölfel Germany
Benjamin Schwalm relative to Alicia Baumgartner Austria Alicia Baumgartner's profile →
Citations per field
00.5×1.5×
Alicia Baumgartner · 1×
Citations per year

Countries citing papers authored by Benjamin Schwalm

Since Specialization
Citations

This map shows the geographic impact of Benjamin Schwalm's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Schwalm with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Schwalm more than expected).

Fields of papers citing papers by Benjamin Schwalm

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Schwalm. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Schwalm. The network helps show where Benjamin Schwalm may publish in the future.

Co-authors

The 25 scholars most cited alongside Benjamin Schwalm, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Benjamin Schwalm Line = papers co-authored together Benjamin Schwalm links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 201733
2 20226
3 20224
4 20251
5 20201
6 20241

About Benjamin Schwalm

Benjamin Schwalm is a scholar working on Genetics, Molecular Biology, Neurology, Ophthalmology and Oncology, having authored 6 papers that have together received 46 indexed citations. Recurring topics across this work include Glioma Diagnosis and Treatment (3 papers), Hedgehog Signaling Pathway Studies (2 papers), Genomics and Chromatin Dynamics (1 paper), Computational Drug Discovery Methods (1 paper), Ocular Oncology and Treatments (1 paper), CAR-T cell therapy research (1 paper), Chromatin Remodeling and Cancer (1 paper) and Neuroblastoma Research and Treatments (1 paper). The work is most often cited by research in Genetics (16 citations), Neurology (8 citations), Biotechnology (5 citations), Biophysics (2 citations) and Molecular Biology (21 citations). Benjamin Schwalm has collaborated with scholars based in Germany, Netherlands and United States. Frequent co-authors include Marcel Kool, Jennifer A. Chan, Andrey Korshunov, Till Milde, Sebastian Brabetz, Christel Herold‐Mende, Christin Schmidt, Norman Mack, Stefan M. Pfister and Florian Selt. Their work appears in journals such as Neuro-Oncology, International Journal of Molecular Sciences, Journal of Clinical Investigation and Neuro-Oncology Advances.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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