Benjamin Amidon
Impact in
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- Cellular transport and secretion
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- Ubiquitin and proteasome pathways
- Protein Degradation and Inhibitors
- Lipid Membrane Structure and Behavior
- Cancer-related gene regulation
Papers in
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- Ubiquitin and proteasome pathways 3
- DNA Repair Mechanisms 1
- Gene expression and cancer classification 1
- Mitochondrial Function and Pathology 1
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- Hippo pathway signaling and YAP/TAZ 2
- Endoplasmic Reticulum Stress and Disease 1
- Co-authors
- Moseley Waite (2 shared papers)Michael A. Milhollen (1 shared paper)Peter G. Smith (1 shared paper)Teresa A. Soucy (1 shared paper)Petter O. Veiby (1 shared paper)Usha Narayanan (1 shared paper)Amy Brown (1 shared paper)Jeffrey D. Schmitt (1 shared paper)
- Journals
- Cancer Research (3 papers)Molecular Cancer Therapeutics (2 papers)Biochemistry (2 papers)BioTechniques (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- United StatesJapan
In The Last Decade
Benjamin Amidon
8 papers receiving 283 citations
Peers
Comparison fields: 5 of 42
- Cell Biology 78
- Molecular Biology 219
- Oncology 77
- Biochemistry 19
- Physiology 37
Countries citing papers authored by Benjamin Amidon
This map shows the geographic impact of Benjamin Amidon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Amidon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Amidon more than expected).
Fields of papers citing papers by Benjamin Amidon
This network shows the impact of papers produced by Benjamin Amidon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Amidon. The network helps show where Benjamin Amidon may publish in the future.
Co-authors
The 25 scholars most cited alongside Benjamin Amidon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 130 | |
| 2 | 1996 | 52 | |
| 3 | 1995 | 33 | |
| 4 | 2012 | 31 | |
| 5 | 2012 | 16 | |
| 6 | 2022 | 14 | |
| 7 | 2021 | 7 | |
| 8 | 2017 | 1 | |
| 9 | 2004 | 0 |
About Benjamin Amidon
Benjamin Amidon is a scholar working on Molecular Biology, Cell Biology, Oncology, Biochemistry and Surgery, having authored 9 papers that have together received 284 indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (3 papers), Lipid metabolism and biosynthesis (2 papers), Cancer-related Molecular Pathways (2 papers), Hippo pathway signaling and YAP/TAZ (2 papers), DNA Repair Mechanisms (1 paper), Gene expression and cancer classification (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper) and Mitochondrial Function and Pathology (1 paper). The work is most often cited by research in Cell Biology (78 citations), Molecular Biology (219 citations), Oncology (77 citations), Biochemistry (19 citations) and Physiology (37 citations). Benjamin Amidon has collaborated with scholars based in United States and Japan. Frequent co-authors include Moseley Waite, Michael A. Milhollen, Peter G. Smith, Teresa A. Soucy, Petter O. Veiby, Usha Narayanan, Amy Brown, Jeffrey D. Schmitt, Tom Thurén and Lynn King. Their work appears in journals such as Cancer Research, Molecular Cancer Therapeutics, Biochemistry, BioTechniques and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.