Béla Kis

105 papers receiving 2.7k citations

Peers

Béla Kis
Comparison fields: 5 of 118
  • Developmental Neuroscience 309
  • Cellular and Molecular Neuroscience 713
  • Neurology 304
  • Pathology and Forensic Medicine 631
  • Biochemistry 199
Replace Mark A. Lovell with:
Mark A. Lovell United States
Kazufumi Kimura Japan
Suzanne de la Monte United States
Weilin Xu China
Louis G. D’Alecy United States
Nobuo Noshita United States
Paul D. Drew United States
Kouichi Tanonaka Japan
Cinzia Dello Russo Italy
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Citations per year

Countries citing papers authored by Béla Kis

Since Specialization
Citations

This map shows the geographic impact of Béla Kis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Béla Kis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Béla Kis more than expected).

Fields of papers citing papers by Béla Kis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Béla Kis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Béla Kis. The network helps show where Béla Kis may publish in the future.

Co-authors

The 25 scholars most cited alongside Béla Kis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Béla Kis Line = papers co-authored together Béla Kis links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 110 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005174
2 2003125
3 2004122
4 201185
5 200278
6 200175
7 200370
8 200369
9 200367
10 200464
11 200264
12 200761
13 200561
14 200461
15 200560
16 201857
17 200556
18 200753
19 200252
20 200651

About Béla Kis

Béla Kis is a scholar working on Cellular and Molecular Neuroscience, Surgery, Pathology and Forensic Medicine, Molecular Biology and Pulmonary and Respiratory Medicine, having authored 110 papers that have together received 2.7k indexed citations. Recurring topics across this work include Cardiac Ischemia and Reperfusion (19 papers), Neuroscience and Neuropharmacology Research (18 papers), Hepatocellular Carcinoma Treatment and Prognosis (15 papers), Neuropeptides and Animal Physiology (15 papers), Anesthesia and Neurotoxicity Research (14 papers), Receptor Mechanisms and Signaling (10 papers), Eicosanoids and Hypertension Pharmacology (10 papers) and Nitric Oxide and Endothelin Effects (9 papers). The work is most often cited by research in Developmental Neuroscience (309 citations), Cellular and Molecular Neuroscience (713 citations), Neurology (304 citations), Pathology and Forensic Medicine (631 citations) and Biochemistry (199 citations). Béla Kis has collaborated with scholars based in United States, Hungary and Japan. Frequent co-authors include David W. Busija, James A. Snipes, Ferenc Bari, Takashi Horiguchi, Krisztina Nagy, Tamás Gáspár, Ferenc Domoki, Nishadi Rajapakse, Zsombor Lacza and Katsuyoshi Shimizu. Their work appears in journals such as Journal of Vascular and Interventional Radiology, Brain Research, Journal of Clinical Oncology, Journal of Cerebral Blood Flow & Metabolism and Journal of Neurochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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